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164篇 您的检索式：作者名="Rockenstein"

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	题名	作者	年代	出处	被引量
1	Altered expression of glutamate transporters under hypoxic conditions in vitro显示文摘	Hsu L Rockenstein E Mallory M	2001	J Neurosci Res2001，64，：	1
2	Dopaminergic loss andinclusion body formation in alpha-synuclein mice: implications for neurodegenerative disorders显示文摘	Masliah E Rockenstein E Veinbergs I	2000	Science2000，287，5456：	1
3	Beta-synuclein inhibits alpha-synuclein aggregation: a possible role as an anti-parkinsonian factor显示文摘	Hashimoto M Rockenstein E Mante M	2001	Neuron2001，32，2：	1
4	Neuroprotective effects of regulators of the glycogen synthase kinase-3beta sig- naling pathway in a transgenic model of Alzheimer's disease are associated with reduced amyloid precursor protein phospho- rylation显示文摘	Rockenstein E Torrance M Adame A	2007	J Neurosci2007，27，8：	1
5	Neprilysin gene transfer reduces human amypoid pathology in transgenic mice显示文摘	Robert A Marr Edward Rockenstein Atish Mukherjee	2003	J Neuroscience2003，23，6：	1
6	β-Amyloid peptides enhance α-synuelein accumulation and neuronal deficits in atransgenic mouse model linking Alzheimer's disease and Parkinson's disease 显示文摘	Masliah E Rockenstein E Veinbergs I	2001	Neurobiology2001，98，12：	1
7	A blood-brain barrier(BBB) disrupter is also a potent alpha-synuclein(alpha-syn) aggregation inhibitor:a novel dual mechanism of mannitol for the treatment of Parkinson disease(PD)显示文摘	Shaltiel-Karyo R Frenkel-Pinter M Rockenstein E		0，，24：	1
8	Effects of alpha-synuclein immunization in a mouse model of Parkinson's disease 显示文摘	Masliah E Rockenstein E Adame A	2005	Neuron2005，46，6：	1
9	Targeting BACE1 with siRNAs ameliorates Alzheimer disease neuropathology in a transgenic model 显示文摘	Singer O Marr RA Rockenstein E	2005	Nat Neurosci2005，8，10：	1
10	β-Amyloid peptides enhance α-synuclein accumulation and neuronal deficits in a transgenic mouse model linking Alzheimers disease and Parkinsons disease显示文摘	Masliah E Rockenstein E Veinbergs I	2001	Proc Natl Acad Sci USA2001，98，12：	1
11	Targeting BACE1 with siRNAs ameliorates Alzheimer disease neuropathology in a transgenic model显示文摘	Singer O Marr R A Rockenstein E	2005	Nat Neurosci2005，8，10：	1
12	β-synuclein inhibits α-synuclein aggregation: A possible role as an anti-Parkinsonian factor显示文摘	Hashimoto M Rockenstein E Mante M	2001	Neuron2001，32，2：	1
13	Altered expression of the synuclein family mRNA in Lewy body and Alzheimer' s disease显示文摘	Rockenstein E Hansen LA Mallory M	2001	Brain Res2001，914，12：	1
14	Neprilysin gene transfer reduces human arnyloid pathology in transgenic mice显示文摘	Marr R A Rockenstein E Mukherjee A	2003	Journal of Neuroscience2003，23，6：	1
15	Central nervous system da- mage produced by expression of the HIV-1 coat protein gpl20 in trans- genic mice显示文摘	Toggas SM Masliah E Rockenstein EM	1994	Nature1994，367，6459：	1
16	Astroglial Expression of Human α 1 -Antichymotrypsin Enhances Alzheimer-like Pathology in Amyloid Protein Precursor Transgenic Mice显示文摘	Lennart Mucke Gui-Qiu Yu Lisa McConlogue Edward M. Rockenstein Carmela R. Abraham Eliezer Masliah	2000	The American Journal of Pathology2000，，6：	1
17	Dopaminergic loss and inclusion body formation in alpha-synuclein mice:implications for neurodegenerative disorders显示文摘	Masliah E Rockenstein E Veinbergs I	2000	Science2000，287，5456：	1
18	Dopaminergic loss and inclusion body formation in alpha-synuclein mice:implications for neurodegenerative disorders显示文摘	Masliah E Rockenstein E Veinbergs I	2000	Science2000，287，：	1
19	beta-Synuclein inhibits alpha-synuclein aggregation:a possible role as an anti-parkinsonian factor显示文摘	Hashimoto M Rockenstein E Mante M	2001	Neuron2001，32，2：	1
20	Dopaminergic loss and inclusion body formation in a-Synuclein mice: implications for neurodegenerative disorders显示文摘	Masliah E Rockenstein E Veinbergs I	2000	Science2000，287，5456：	1

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