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| 1 | Quantitative analysis of brain perfusion SPECT in Alzheimer's disease using a fully automated regional cerebral blood flow quantification software,3DSRT显示文摘 | Seiju Kobayashi Masaru Tateno Kumiko Utsumi | | 0，，01： | 1 |
| 2 | A Novel Vascular Modulator, Hepatocyte Growth Factor (HGF), as a Potential Index of the Severity of Hypertension显示文摘 | Shigefumi Nakamura Atsushi Moriguchi Ryuichi Morishita Motokuni Aoki Yoshikage Yo Shin-ichiro Hayashi Nobuaki Nakano Tomohiro Katsuya Sachiko Nakata Seiju Takami Kunio Matsumoto Toshikazu Nakamura Jitsuo Higaki Toshio Ogihara | 1998 | Biochemical and Biophysical Research Communications1998，，1： | 1 |
| 3 | Human G-protein β3 subunit variant is associated with serum potassium and total cholesterol levels but not with blood pressure显示文摘 | Kazuhiko Ishikawa Yutaka Imai Tomohiro Katsuya Takayoshi Ohkubo Ichiro Tsuji Kenichi Nagai Seiju Takami Yukiko Nakata Hiroshi Satoh Shigeru Hisamichi Jitsuo Higaki Toshio Ogihara | 2000 | American Journal of Hypertension2000，，2： | 1 |
| 4 | Polymorphism of the Apolipoprotein E and Angiotensin-Converting Enzyme Genes in Japanese Subjects With Silent Myocardial Ischemia显示文摘 | Yukiko Nakata Tomohiro Katsuya Hiromi Rakugi Seiju Takami Mitsuru Ohishi Kouzin Kamino Jitsuo Higaki Yoshikatsu Tabuchi Yuichi Kumahara Tetsuro Miki Toshio Ogihara | 1996 | Hypertension1996，，6： | 1 |
| 5 | New Cylindrocladium diseases of strawberry and coral bells caused by Cylindrocladium canadense 显示文摘 | Yuuri Hirooka Seiju Ishikawa Jun Takeuchi | 2009 | J Gen Plant Pathol2009，75，： | 1 |
| 6 | Pneumonia and seizures due to hypereosinophilic syndrome—organ damage and eosinophilia without synchronisation:A case report显示文摘BACKGROUND Hypereosinophilic syndrome(HES)is a condition characterized by increased eosinophil proliferation in the bone marrow,as well as tissue eosinophilia,often causing organ damage.The cause of the disease is unknown.Initial symptoms include fatigue,cough,shortness of breath,myalgia,angioedema,fever,and pneumonia.In addition to the respiratory symptoms,damage to the central nervous system can lead to severe seizures.Here,we report a case with pneumonia and complex partial seizures secondary to HES.CASE SUMMARY A 94-year-old woman was admitted to our hospital for heart failure and bloody stools.After admission,she also showed symptoms of pneumonia.Non-contrast computed tomography of the chest showed pleural effusion and infiltrative shadows.Lower gastrointestinal endoscopy showed multiple ulcers in the sigmoid colon.Blood analyses showed marked eosinophilia(eosinophils 1760/mm3,total leukocytes 6850/mm3).Initial treatment with furosemide 20 mg/d and prednisolone 25 mg/d relieved these symptoms.However,the patient subsequently experienced localised epileptic seizures characterized by bilateral eyelid twitching and eyes rolling upwards,without generalized convulsions,and respiratory arrest occurred.Electroencephalography showed spikes and waves.Non-contrast magnetic resonance imaging of the brain showed extensive periventricular hyperintensity.With administration of levetiracetam 1000 mg/d the epileptic seizures disappeared.However,the patient’s consciousness remained impaired,and her pneumonia worsened again.Two weeks later,she died of pneumonia.CONCLUSION HES symptoms are variable and atypical,and the level and timing of eosinophilia and organ damage are often discordant. | Tetsuro Ishida Tomonori Murayama Seiju Kobayashi | 2022 | World Journal of Clinical Cases2022，10，18： | 1 |
| 7 | Current research of idiopathic normal pressure hydrocephalus:Pathogenesis,diagnosis and treatment显示文摘Idiopathic normal pressure hydrocephalus(iNPH)is caused by impaired cerebrospinal fluid absorption in the elderly;it is a surgically treatable form of dementia.Gait disturbance,dementia,and urinary incontinence are the triad of signs for iNPH.In addition to these clinical findings,imaging studies show characteristic ventricular enlargement.High Evans Index and‘disproportionately enlarged subarachnoid hydrocephalus’are other well-known imaging findings of iNPH.If the tap test shows improved symptoms,shunt surgery is performed.The disease was first described by Hakim and Adams in 1965,followed by the publication of the first,second,and third editions of the guidelines in 2004,2012,and 2020,respectively.Recent studies signal the glymphatic system and classical cerebrospinal fluid(CSF)absorption from the dural lymphatics as aetiological mechanisms of CSF retention.Research is also underway on imaging test and biomarker developments for more precise diagnosis,shunting technique options with fewer sequelae and complications,and the influence of genetics.Particularly,the newly introduced‘suspected iNPH’in the third edition of the guidelines may be useful for earlier diagnosis.However,less well-studied areas remain,such as pharmacotherapy in non-operative indications and neurological findings other than the triadic signs.This review briefly presents previous research on these and future issues. | Tetsuro Ishida Tomonori Murayama Seiju Kobayashi | 2023 | World Journal of Clinical Cases2023，11，16： | 0 |
| 8 | Longitudinal observation of ten family members with idiopathic basal ganglia calcification: A case report显示文摘BACKGROUND Familial idiopathic basal ganglia calcification (FIBGC) is a rare autosomal dominant disorder that causes bilateral calcification of the basal ganglia and/or cerebellar dentate nucleus, among other locations. CASE SUMMARY The aim of this study is to report 10 cases of FIBGC observed in a single family. Seven patients showed calcification on their computed tomography scan, and all of these patients carried the SLC20A2 mutation. However, individuals without the mutation did not show calcification. Three patients among the 7 with calcification were symptomatic, while the remaining 4 patients were asymptomatic. Additionally, we longitudinally observed 10 subjects for ten years. In this paper, we mainly focus on the clinical course and neuroradiological findings in the proband and her son.CONCLUSION The accumulation of more case reports and further studies related to the manifestation of FIBGC are needed. | Seiju Kobayashi Kumiko Utsumi Masaru Tateno Tomo Iwamoto Tomonori Murayama Hitoshi Sohma Wataru Ukai Eri Hashimoto Chiaki Kawanishi | 2019 | World Journal of Clinical Cases2019，7，12： | 0 |
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