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9篇 您的检索式：作者名="DiLISA F"

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		题名	作者	年代	出处	被引量
	1	Opening of the mitochondrial permeability transition pore causes depletion of mitochondrial and cytosolic NAD^+ and is a causative event in the death of myocytes in postischemic reperfusion of the heart显示文摘	DILISA F MENABO R CANTON M	2001	J Biol Chem2001，276，4：	1
	2	Quantitation of the effect of L-carnitine on the levels of acid-soluble shortchain acyl-CoA and CoASH in rat heart and liver mitochondria显示文摘	Lysiak W Lilly K DiLisa F	1988	J BioI Chern1988，263，3：	1
	3	Mitochondrial pathways for ROS formation and myocardial injury:the rel-evance of p66Shc and monoamine oxidase显示文摘	Dilisa F Kaludercic N Carpi A	2009	Basic Res Cardiol2009，104，2：	1
	4	Specific degradation of troponin T and I by mu - calpain and its modulation by substratephosphorylation显示文摘	Dilisa F De tullio R Salamino F	1995	Biochem J1995，308，：	1
	5	Influence of L-carnitine administration on maximal physical exercise显示文摘	Vecchiet L Dilisa F Pieralis G	1990	Euro J Appl Physiol Occup Physiol1990，61，56：	1
	6	Specific degradation of troponin T and I by mu-calpain and its modulation by substrate phosphorylation显示文摘	Detullio K Salamino F	1995	Biochem J1995，308，：	1
	7	Mitochondrial function as a determinant of recovery or death in cell response to injury显示文摘	DiLISA F BERNARDI P	1998	Mol Cell Biochem1998，184，12：	1
	8	Specific degradation of troponin T and I by m-calpain and its modutation by substrate phosophorytation显示文摘	DILISA F DETULLIO K SALAMINO F	1995	Biochem J1995，308，：	1
	9	Postconditioning and protection from reperfusion injury:where do we stand? Position paper from the Working Group of Cellular Biology of the Heart of the European Society of Cardiology显示文摘	Ovize M Baxter GF DiLisa F	2010	Cardiovasc Res2010，87，：	1

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