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20篇 您的检索式:作者名="Kaludercic N"
    题名 作者 年代 出处 被引量
1Modulation of mitochon- drial proteome and improved mitochondrial function by biventricu- lar pacing of dyssynchronous failing hearts 显示文摘Agnetti G Kaludercic N Kane LA 2010Circ Cardiovasc Genet2010,3,:1
2Monoamine oxidase A-mediated enhanced catabolism of norepinephrine contributes to adverse remodeling and pump failure in hearts with pressure overload 显示文摘Kaludercic N Takimoto E Nagayama T 2010CircRes2010,106,1:1
3Modulation of mitochon- drial proteome and improved mitoehondrial function by biventricular pacing of dyssynchronous failing hearts显示文摘Agnetti G Kaludercic N Kane LA et 81 2010Circ Cardiovasc Genet2010,3,:1
4Reactive oxygen species and redox compartnmntalization显示文摘KALUDERCIC N DESHWAL S DI LISA F 2014Front Physiol2014,5,:1
5Beta(2)-Adrenoceptors,NADPH oxidase,ROS and p38MAPK:another ‘radical’ road to heart failure?显示文摘DI LISA F KALUDERCIC N PAOLOCCI N 2011Br JPharmacol2011,162,5:1
6Modulation of mitochondrial proteome and improved mitochondrial function by biventricular pacing of dyssynchronous failing hearts显示文摘Agnetti G Kaludercic N Kane L A 2010Circ Cardiovasc Genet2010,3,1:1
7Modulation of mito- chondrial proteome and improved mitoehondrial function by biventrieular pacing of dyssynchronous failing hearts 显示文摘Aguetti G Kaludercic N Kane LA 2010Circ Cardiovasc Genet2010,3,1:1
8Monoamine oxidases (MAO) in the pathogenesis of heart failure and ischemia/reperfusion injury 显示文摘Kaludercic N Carpi A Menabb R 2011Biochim Biophys Acta2011,1813,7:1
9Inhibiting metalloproteases with PD 166793 in heart failure:pact on cardiac remodeling and beyond显示文摘Kaludercic N Lindsey ML Tavazzi B 2008Cardiovasc Ther2008,269,1:1
10Mitochondria and vascular pa- thology显示文摘di Lisa F Kaludercic N Cappi A 2009Pharmacological Reports2009,61,1:1
11Mitochondria and vascu- lar pathology显示文摘Di Lisa F Kaludercic N Carpi A 2009Pharmacol Rep2009,61,1:1
12Inhibiting metalloproteases with PD 166793 in heart failure: im- pact on cardiac remodeling and beyond 显示文摘Kaludercic N Lindsey ML Tavazzi B 2008Cardiovasc Ther2008,26,1:1
13Mitochondrial pathways for ROS formation and myocardial injury:the rel-evance of p66Shc and monoamine oxidase显示文摘Dilisa F Kaludercic N Carpi A 2009Basic Res Cardiol2009,104,2:1
14Monoamine oxi-dases(MAO)in the pathogenesis of heart failure and is-chemia/reperfusion injury显示文摘Kaludercic N Carpi A MenabòR 2011Biochim Biophys Acta2011,1813,7:1
15Monoamine oxidases(MAO)inthe pathogenesis of heart failure and ischemia/reperfusion injury显示文摘Kaludercic N Carpi A MenabòR 2011Biochim Biophys Acta2011,1813,7:1
16beta-Adrenoceptors,NADPH oxidase,ROS and p38 MAPK:another 'radical' road to heart failure Br显示文摘Di Lisa F Kaludercic N Paolocci N 0,,:1
17Mitochon-drial pathways for ROS formation and myocardial injury: the relevance of p66Shc and monoamine oxi- dase显示文摘Lisa F Di Kaludercic N Carpi A 2009Basic Res Cardiol2009,104,2:1
18Monoamine oxidase A mediated enhanced catabolism of norepinephrine contributes to adverse remodeling and pump failure in hearts with pressure overload显示文摘Kaludercic N Takimoto E Nagayama T 2010Cric Res2010,106,1:1
19β2-Adrenoceptors,NADPH oxidase,ROS and p38 MAPK:another‘radical’road to heart failure显示文摘Di Lisa F Kaludercic N Paolocci N 2011Br J Pharmacol2011,162,5:1
20β-Adrenoceptors,NADPH oxidase,ROS and p38 MAPK:another ' radical' road to heart failure?显示文摘Di Lisa F Kaludercic N Paolocci N 2011Br J Pharmacol2011,162,5:1
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