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18篇 您的检索式:作者名="David Sachs"
    题名 作者 年代 出处 被引量
13-Draw: A tool for designing 3D shapes 显示文摘 Andrew Roberts David Stoops 1991IEEE Computer Graphics and Applications1991,11,6:1
2Estimating radiation-induced cancer risks at very low doses: rationale for using a linear no-threshold approach显示文摘David J. Brenner Rainer K. Sachs 2006Radiation and Environmental Biophysics2006,,4:1
3The role of internal urease in acid resistance of Helicobacter pylori显示文摘David R. Scott David Weeks Charlie Hong Stefan Postius Klaus Melchers George Sachs 1998Gastroenterology1998,,1:1
4Automated Assignment of SCOP and CATH Protein Structure Classifications From FSSP Scores显示文摘Gad Getz Michele Vendruscolo David Sachs 2002Proteins2002,46,:1
5A Fallacy in Plato's Republic显示文摘David Sachs 1963The Philosophical Review1963,72,2:1
6Characterization of an expressed pig class Ⅱ gene homologous to HlA-DQA 显示文摘Hirsch G David H Sachs E 1990Immunogenetics1990,31,:1
7Mixed lymphohaemopoietic chimerism and graft-ver suslymphoma effects after non-myeloablative therapy and HLA-mismatched bone-marrow transplantation显示文摘Megan Sykes Frederic Preffer Steven McAfee Susan L Saidman Dina Weymouth David M Andrews Christine Colby Robert Sackstein David H Sachs Thomas R Spitzer 1999The Lancet1999,,9166:1
8Injection of Allogeneic Bone Marrow Cells into the Portal Vein of Swine in Utero显示文摘J.Peter Rubin Sheldon R. Cober Peter E.M. Butler Mark A. Randolph G.Scott Gazelle Francisco L. Ierino David H. Sachs W.P.Andrew Lee 2000Journal of Surgical Research2000,,2:1
9A Fallacy in Plato's Republic 显示文摘David Sachs 1963The Philosophical Review1963,72,2:1
10Alteration of gene expression during environmental stressing plants 显示文摘SACHS M M DAVID H T U 1986Annu Rev Plant Physiol1986,37,:1
11Parathyroid hormone and arterial dysfunction in the m ulti‐ e thnic s tudy of a therosclerosis显示文摘Cortney Bosworth Michael C. Sachs Daniel Duprez Andrew N. Hoofnagle Joachim H. Ix David R. Jacobs Carmen A. Peralta David S. Siscovick Bryan Kestenbaum Ian H. Boer 2013Clin Endocrinol2013,,3:1
12Gastric infection by Helicobacter pylori显示文摘George Sachs Yi Wen David R. Scott 2009Current Gastroenterology Reports2009,,6:1
133-Draw: A tool for designing 3D shapes显示文摘Emanuel Sachs Andrew Roberts and David Stoops 1991IEEE Computer Graphics and Applications1991,11,6:1
1425-Hydroxyvitamin D and Parathyroid Hormone Are Not Associated With Carotid Intima-Media Thickness or Plaque in the Multi-Ethnic Study of Atherosclerosis显示文摘Marc Blondon Michael Sachs Andrew N. Hoofnagle Joachim H. Ix Erin D. Michos Claudia Korcarz Adam D. Gepner David S. Siscovick Joel D. Kaufman James H. Stein Bryan Kestenbaum Ian H. de Boer 2013Arteriosclerosis Thrombosis and Vascular Biology2013,,11:1
15Alteration of gene expression during environmental stress in plant显示文摘SACHS M M David T H 1986Physiol Plant1986,37,:1
16Alteration of gene expression during environmental stress in plant显示文摘Sachs M M David H T U 1998Annu Rew Plant Physiol1998,37,:1
17Factors associated with success of telaprevir-and boceprevir-based triple therapy for hepatitis C virus infection显示文摘AIM To evaluate new therapies for hepatitis C virus(HCV), data about real-world outcomes are needed.METHODS Outcomes of 223 patients with genotype 1 HCV who started telaprevir-or boceprevir-based triple therapy(May 2011-March 2012) at the Mount Sinai Medical Center were analyzed. Human immunodeficiency viruspositive patients and patients who received a liver transplant were excluded. Factors associated with sustained virological response(SVR24) and relapse were analyzed by univariable and multivariable logistic regression as well as classification and regression trees. Fast virological response(FVR) was defined as undetectable HCV RNA at week-4(telaprevir) or week-8(boceprevir). RESULTS The median age was 57 years, 18% were black, 44% had advanced fibrosis/cirrhosis(FIB-4 ≥ 3.25). Only 42%(94/223) of patients achieved SVR24 on an intention-totreat basis. In a model that included platelets, SVR24 was associated with white race [odds ratio(OR) = 5.92, 95% confidence interval(CI): 2.34-14.96], HCV sub-genotype 1b(OR = 2.81, 95%CI: 1.45-5.44), platelet count(OR = 1.10, per x 104 cells/μL, 95%CI: 1.05-1.16), and IL28 B CC genotype(OR = 3.54, 95%CI: 1.19-10.53). Platelet counts > 135 x 103/μL were the strongest predictor of SVR by classification and regression tree. Relapse occurred in 25%(27/104) of patients with an end-oftreatment response and was associated with non-FVR(OR = 4.77, 95%CI: 1.68-13.56), HCV sub-genotype 1a(OR = 5.20; 95%CI: 1.40-18.97), and FIB-4 ≥ 3.25(OR = 2.77; 95%CI: 1.07-7.22). CONCLUSION The SVR rate was 42% with telaprevir-or boceprevirbased triple therapy in real-world practice. Low platelets and advanced fibrosis were associated with treatment failure and relapse.Kian Bichoupan Neeta Tandon Valerie Martel-Laferriere Neal M Patel David Sachs Michel Ng Emily A Schonfeld Alexis Pappas James Crismale Alicia Stivala Viktoriya Khaitova Donald Gardenier Michael Linderman William Olson Ponni V Perumalswami Thomas D Schiano Joseph A Odin Lawrence U Liu Douglas T Dieterich Andrea D Branch 2017World Journal of Hepatology2017,9,11:0
18移植的耐受性David H Sachs 耿稚江 1994国外医学情报1994,,23:0
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