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| 1 | The assessment and management of chronic cough in children according to the British Thoracic Society guidelines : descriptive, prospective, clinical trial 显示文摘 | Usta Guc B Asilsoy S Durmaz C | 2014 | Clin Respir J2014，8，3： | 1 |
| 2 | Evidence that aminoguanidine inhibits endotoxin - induced bacterial translocation显示文摘 | Agalar B K C Guc M O Sayek I | 1998 | Br J Surg1998，85，： | 1 |
| 3 | Towards trajectory anonymization:a generalization-based approach显示文摘 | M E Nergiz M Atzori Y Saygin B Guc | 2009 | Transactions on Data Privacy2009，2，1： | 1 |
| 4 | The assessment and manage ment of chronic cough in children according to the British Thoracic Society guidelines: descriptive, prospective, clinical trial显示文摘 | Usta Guc B Asilsoy S Durmaz C | 2014 | Clin Respir J2014，8，3： | 1 |
| 5 | The assessment and management of chronic cough in children according to the British Thoracic Society guidelines:descriptive,prospective,clinical trial显示文摘 | Usta Guc B Asilsoy S Durmaz C | 2014 | Clin Respir J2014，8，3： | 1 |
| 6 | The assessment and management of chronic cough in children according to the British Thoracic Society guidelines:descriptive,prospective,clinical trial显示文摘 | Usta Guc B Asilsoy S Durmaz C | 2014 | Clin Respir J2014，8，3： | 1 |
| 7 | PISO Algorithm for Un- steady Flow Field 显示文摘 | Anaheim Calif USA Wang T GuC G 'fang B | 2003 | Journal of Hydrodynamics2003，18，： | 1 |
| 8 | The assessment and managemerit of chronic cough in children according to the British Thoracic Society guidelines: descriptive, prospective, clinical trial 显示文摘 | Usta Guc B Asilsoy S Durmaz C | 2014 | Clin Respir J2014，8，3： | 1 |
| 9 | 内皮素和一氧化氮对脓毒性休克实验模型中器官损伤,肠系膜缺血和存活的影响(英文)显示文摘The development of potent drugs to treat cardiopulmonary failure in sepsis, such as antibiotics and new immunomodulatory therapeutic approaches have not prevented sepsis from being a major health problem. Dysfunction of the vascular endothelium is an early event in septic shock. The recognition of endothelium-derived substances, such as nitric oxide and endothelin, important mediators of systemic inflammatory response syndrome,led to the proposal that pharmacological inhibition of nitric oxide and endothelin production could represent a useful strategy in the treatment of septic shock. Splanchnic ischemia and translocation of endotoxin from the gut to the circulation contributes significantly to the high mortality rate in sepsis-related syndromes. This vasoconstriction in the splanchnic circulation can be partially blocked by inducible nitric oxide synthase inhibitor aminoguanidine or endothelin receptor antagonist bosentan in experimental models of septic shock. It can be suggested that endothelin and nitric oxide may affect survival. Although septic shock is a highly complex pathophysiological state, the course of septic shock has different phases with different characteristics which need different (special) treatment strategy. The inhibition of nitric oxide production during hyperdynamic,earlier phase of sepsis combined with the blockade of endothelin receptors at a later stage during the hypodynamic,late phase appears to be a novel promising strategy for the therapy of septic shock. The aim of this review is to discuss the role of nitric oxide and endothelin in sepsis and the potential therapeutic implications of blockade of nitric oxide and endothelin as a target in treatment of human septic shock. Briefly the importance of timing of intervention is also emphasized. | Alper B ISKIT Oguz GUC | 2003 | Acta Pharmacologica Sinica2003，24，10： | 1 |
| 10 | Cytomegalo- vires infection in a patient with atypical Kawnsaki disease 显示文摘 | Usta Guc B Cengiz N Yildirim SV | 2008 | J Rheumatol Int2008，28，4： | 1 |
| 11 | Effects of endothelin and nitric oxide on organ injury, mesenteric ischemia, and survival in experimental models of septic shock 显示文摘 | Iskit A B Guc O | 2003 | Acta Pharmacol Sin2003，24，10： | 1 |
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