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13篇 您的检索式：作者名="Weppler SA"

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		题名	作者	年代	出处	被引量
	1	Hypoxia as target for combined modality treatments显示文摘	Weppler SA Koritzinsky M	2002	Eur J Cancer2002，38，2：	1
	2	Combined RNAi-medialed suppression of Rictor and EGFR resulted in completetumor regression in an orthotopic glioblastoma tumor model 显示文摘	Verreault M Weppler SA Stegeman A	2013	PLoS One2013，8，59：	1
	3	Hypoxia as a target for combined modality treatments 显示文摘	Wouters BG Weppler SA Koritzinsky M	2002	Eur J Cancer2002，38，2：	1
	4	Expression of EGFR variant vIII promotes both radiation resistance and hypoxia tolerance显示文摘	Weppler SA Li Y Dubois L	2007	Radiother Oncol2007，83，3：	1
	5	Expression of EGFR variant vⅢ promotes both radiation resistance and hypoxia tolerance显示文摘	Weppler SA Li Y Dubois L	2007	Radiother Oncol2007，83，2：	1
	6	The combination of gefitinib and RAD001 inhibits growth of HER2 overex- pressing breast cancer cells and tumors ir- respective of trastuzumab sensitivity 显示文摘	Dragowska WH Weppler SA Qadir MA	2011	BMC Cancer2011，11，：	1
	7	Hypoxia as a target for combined modality treatments显示文摘	Wouters BG Weppler SA Koritzinsky M	2002	Eur J Cancer2002，38，2：	1
	8	The combination of gefitinib and RAD001 inhibits growth of HER2 overexpressing breast cancer cells and tumors irrespective of trastuzumab sensi- tivity显示文摘	Dragowska WH Weppler SA Qadir MA	2011	BMC Cancer2011，11，：	1
	9	Expression of EGFR variant vIHpromotes both radiation resistance and hypoxia tolerance 显示文摘	Weppler SA Li Y Dubois L	2007	Ra-diother Oncol2007，83，2：	1
	10	Induction of autophagy is an early response to gefitinib and a potential therapeutic target in breast cancer显示文摘	Dragowska WH Weppler SA Wang JC	2013	PLoS One2013，8，76：	1
	11	Induction of autophagy is an early response to gefitinib and a poten- tial therapeutic target in breast cancer显示文摘	Dragowska WH Weppler SA Wang JC	2013	PLoS One2013，8，76：	1
	12	Expression of egfr variant viii promotes both radiation resistance and hypoxia tolerance 显示文摘	Weppler SA Li Y Dubois L	2007	Radiother Oncol2007，83，3：	1
	13	Induction of autophagy is an early response to gefitinib and a potential therapeutic target in breast cancer显示文摘	Dragowska WH Weppler SA Wang JC	2013	PLoS One2013，8，76：	1

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