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43篇 您的检索式:作者名="WILMEN A"
    题名 作者 年代 出处 被引量
1In vitro and in vivo studies of the novel antithrombotic agent BAY 59-7939--an oral, direct Factor Xa inhibitor显示文摘Perzborn E Strassburger J Wilmen A 2005J Thromb Haemost2005,3,3:1
2In vitro and in vivo studies of the novel antithrombotic agent BAY 59-7939-an oral, direct factor Xa inhibitor 显示文摘Perzborn E Strassburger J Wilmen A 2005J Thromb Haemost2005,3,3:1
3In vitro and in vivo studies of the novel antithrombotic agent BAY 597939-an oral, direct factor Xa inhibitor显示文摘PERZBORN E STRASSBURGER J WILMEN A 2005J Thromb Haemost2005,3,3:1
4In vitro and in vivo studies of the novel antithrombotic agent BAY 59-7939 an oral, direct factor X a inhibitor 显示文摘Perzborn E Strassburger J Wilmen A 2005J Thromb Haemost2005,3,3:1
5In vitro and in vivo studies of the novel antithrombotic agent BAY 59-7939- an oral , direct factor Xa inhibitor显示文摘PERZBORN E STRASSBURGER J WILMEN A 2005J Thromb Haemost2005,3,3:1
6Roles of TNF-related apoptosis-inducing ligand in experimental autoimmune encephalomyelitis显示文摘Hilliard B Wilmen A Seidel C 2001J Immunol2001,166,2:1
7Roles of TNF-related apoptosis-inducing ligand in experimental autoimmune encephalomyelitis显示文摘 Wilmen A Seidel C 2001J Immunol2001,166,2:1
8In vitro and in vivo, studies of the novel antithrombotic agent BAY 59 - 7939- an o- ral,direct factor X a inhibitor显示文摘Perzborn E Strassburger J Wilmen A 2005J Thromb Haemost2005,3,3:1
9Roles of TNF-related apoptosis-inducing ligand in experimental autommune encephalomyelitis显示文摘Hilliard B Wilmen A Seidel C 2001J Immunol2001,166,2:1
10Five out of six tryptophan residues in the N-terminal extracellular domain of the rat GLP-1 receptor are essential for its ability to bind GLP-1 显示文摘WILMEN A GOKE B VAN EYLL B 1997Peptides1997,18,2:1
11In vitro and in vivo studies of the novel antithrombotic agent BAY 59-7939 an oral,direct factor Xa inhibitor显示文摘Perzbom E Strassburger J Wilmen A 2005J Thromb Haemost2005,3,3:1
12Exchange of W39 by A within the N-teminal extrcellular domain of the GLP-1 receptor results in a loss of receptor function 显示文摘VAN EYLL B GOKE B WILMEN A 1996Peptides1996,17,:1
13In vitro and in vivo studies of the novel antithrombotic agent BAY 59-7939 - an oral, direct Factor Xa inhibitor显示文摘Perzborn E Strassburger J Wilmen A 2005J Thromb Haemost2005,3,3:1
14In vitro and in vivo studies of the novel antithrombotic agent BAY 59-7939-an oral,direct Factor Xa inhibitor显示文摘Perzbom E Strassburger J Wilmen A 0,,03:1
15In vitro and in vivo studies of the novel antithrombotic agent BAY 59- 7939-an oral, direct factor Xa inhibitor显示文摘PERZBORN E STRASSBURGER J WILMEN A 2005J Thromb Haemost2005,3,3:1
16In vitro and in vivo studies of the novel antithrombotic agent BAY 59-7939-an oral, direct factor Xa inhibitor显示文摘Perzborn E Strassburger J Wilmen A 2005J Thromb Haemost2005,3,:1
17Five out of six tryptophan residues in the N-terminal extracellu- lar domain of the rat GLP-1 receptor are essential for its ability to bind GLP-1 显示文摘Wilmen A Van Eyll B Goke B 1997Peptides1997,18,2:1
18In vitro and in vivo studies of the novel antithrombotic agent BAY 59-7939-an oral, direct factor Xa inhibitor 显示文摘Perzbom E Strassburger J Wilmen A 2005J Thomb Haemostais2005,3,3:1
19Exchange of W39 by A within the N-terninal extrcellular domain of the GLP-1 receptor results in a loss of receptor function显示文摘B Van Eyll G(o)ke B Wilmen A 0,,:1
20Five out of six tryptophan residues in the N-terminal extracellular domain of the rat GLP-1 receptor are essential for its ability to bind GLP-1显示文摘Wilmen A B Van Eyll G(o)ke B 0,,02:1
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