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52篇 您的检索式：作者名="VOSHOL PJ"

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	题名	作者	年代	出处	被引量
1	ApoAⅤ reduces plasma triglycerides by inhibiting very low density lipoprotein-triglyceride (VLDL-TG) production and stimulating lipoprotein lipase-mediated VLDL-TG hydrolysis显示文摘	Schaap FG Rensen PC Voshol PJ Vrins C Van der Vliet HN Chamuleau RA	2004	J Biol Chem2004，279，27：	1
2	ApoAV reduces plasma triglycerides by inhibiting very low density lipoprotein-triglyceride(VLDL-TG)production and stimulating lipoprotein lipase-mediated VLDL-TG hydrolysis显示文摘	Schaap FG Rensen PC Voshol PJ	2004	J Biol Chem2004，279，27：	1
3	Down-regulation of intestinal scavenger receptor class B,type 1(SRBI) expression in Rodents under conditions of deficient bile delivery to the intestine显示文摘	Voshol PJ Schwarz M Rigotti A	2001	Biochem J2001，356，2：	1
4	Protection from obesity and insu-lin resistance in mice overexpressing human apolipoprotein Cl显示文摘	Jong MC Voshol PJ Muurling M ef al	2001	Dia-betes2001，50，12：	1
5	White adipose tissue: getting nervous显示文摘	Fliers E Kreier F Voshol PJ	2003	J Neuro-endocrinol2003，15，：	1
6	Stimulation of lipogenesis by pharmacological activation of the liver X receptor leads to production of large, triglyeeride-rich very low density lipoprotein particles 显示文摘	Grefhorst A Elzinga BM Voshol PJ	2002	J Biol Chem2002，277，34：	1
7	Chronic PYY3-36 treatment promotes fat oxidation and ameliorates insulin resistance in C57BL6 mice显示文摘	VAN DEN HOEK AM HE1JBOER AC VOSHOL PJ		0，，：	1
8	Stimulation of lipogenesis by pharmacoiogical activation of the liver X receptor leads to production of large, triglyeefide-rich very low density lipoprotein partieles显示文摘	Grefhorst A Elzinga BM Voshol PJ	2002	J Biol Chem2002，277，34：	1
9	Chronic PYY3-36 treatment promotes fat oxidation and ameliorates insulin resistance in C57BL6 mice显示文摘	van den Hoek AM Heijboer AC Voshol PJ	2007	Am J Physiol Endocrinol Metab2007，292，1：	1
10	The Hyplip2 locus causes hypertriglyceridemia by decreased clearance of lriglycerides显示文摘	Moen CJ Tholens AP Voshol PJ	2007	J Lipid Res2007，48，10：	1
11	ApoAV reduces plasma triglycerides by inhibiting very low density lipoprotein-triglyceride (VLDL-TG) production and stimulating lipoprotein lipase-mediated VLDL-TG hydrolysis显示文摘	Schaap FG Rensen PC Voshol PJ	2004	J Biol Chem2004，279，27：	1
12	The Hyplip-locus causes hypertriglyceridemia by decreased clearance of triglyeerides 显示文摘	Moen CJ Tholens AP Voshol PJ etal	2007	Lipid Res2007，48，10：	1
13	Protection From Obesity and Insulin Resistance in Mice Overexpressing Human Apolipoprotein C1 显示文摘	Jong MC Voshol PJ Muurling M	2001	Diabetes2001，50，12：	1
14	The VLDL receptor plays a major role in chylomicron metabolism by enhance LPL -mediated triglyceride hydrolysis显示文摘	Gourdriaan JR Santo SM Voshol PJ	2004	J Lipid Res2004，45，8：	1
15	Stimμlation of lipogene-sis by pharmacological activation of the liver X receptor(LXR)leads to production of large,triglyceride-rich VLDL particles显示文摘	Grefhorst A Elzinga BM Voshol PJ	2002	JBiol Chem2002，277，34：	1
16	ApoAV reduces plasma triglycerides by inhibiting very low density lipoprotein-triglyceride (VLDL-TG) production and stimulating lipoprotein lipasemediated VLDL-TG hydrolysis 显示文摘	Schaap FG Rensen PC Voshol PJ Vrins C van der Vliet HN Chamuleau RA	2004	J Biol Chem2004，279，27：	1
17	AMPK-mediated increase in myocardial long-chain fatty acid uptake critically depends on sarcolemmal CD36显示文摘	Habets DD Coumans WA Voshol PJ	2007	Biochem Biophys Res Commun2007，355，1：	1
18	Chronic PYY3-36 treatment promotes fat oxidation and amel-iorates insulin resistance in C57BL6 mice显示文摘	Vandenhoek AM Heijboer AC Voshol PJ	2007	Am J Physiol Endocrinol Metab2007，292，1：	1
19	ApoAV reduces plasma triglycerides by inhibiting very low density lipoprotein-triglyceride (VLDL-TG) production and stimu-lating lipoprotein lipase-mediated VLDL-TG hydrolysis显示文摘	SCHAAP FG RENSEN PC VOSHOL PJ	2004	J Biol Chem2004，279，27：	1
20	ApoA-V reduces plasma triglycerides by inhibiting very low density lipoprotein-triglyceride (VLDL-TG) production and stimulating lipopmtein lipase-mediated VLDL-TG hydrolysis 显示文摘	Schaap FG Rensen PC Voshol PJ	2004	J Biol Chem2004，279，27：	1

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