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11篇 您的检索式:作者名="Uhrbom"
    题名 作者 年代 出处 被引量
1Dependence of autocrine growth factor stimulation in platelet-derived growth factor-B-induced mouse brain tumor cells显示文摘Uhrbom L Hesselager G Ostman A 2000Int J Cancer2000,85,3:1
2Ink4a-Arf Loss Cooperates with KRas Activation in Astrocytes and Neural Progenitors to Generate Glioblastomas of Various Morphologies Depending on Activated Akt显示文摘Uhrbom L Dai C Celestino JC 2002Cancer Res2002,62,:1
3Ink4a-Arf loss cooperates with KRas activation in astrocytes and neural progenitors to generate glioblastomas of various morpho- logies depending on activated Akt 显示文摘Uhrbom L Dai C Celestino JC et 01 2002Cancer Res2002,62,19:1
4Cell type-specific tumor suppression by Ink4a and Arf in Kras-induced mouse gliomagenesis显示文摘Uhrbom L Kastemar M Johansson FK 2005Cancer Res2005,65,:1
5Complementary effects of platelet-derived growth factor autocrine stimulation and p53 or lnk4a-Arf deletion in a mouse glioma model显示文摘Hesselager G Uhrbom L Westermark B 2003Cancer Res2003,63,:1
6Ink4a-Aft loss cooperates with KRas activation in astroeytes and neural progenitors to generate glioblastomas of various morphologies depending on activated Akt 显示文摘Uhrbom L Dai C Celesfino JC 2002Cancer IRes2002,62,19:1
7Ink4a-Arf loss cooperateswith KRas activation in astrocytes and neural progenitors to generate glioblastomas of various morphologies depending on activated Akt显示文摘Uhrbom L Dai C Celestino JC 2002Cancer Res2002,62,:1
8Dissecting tumor maintenance requirements using bioluminescence imaging of cell proliferation in a mouse glioma model显示文摘Uhrbom L Nerio E Holland EC 0,,11:1
9Complementary effects of platelet-derived growth factor autocrine stimulation and p53 or Ink4a-Arf deletion in a mouse glioma model显示文摘Hesselager G Uhrbom L Westermark B 2003Cancer Res2003,63,15:1
10Ink4a-Arf loss cooperates with KRas activation in astrocytes and neural progenitors to generate glioblastomas of various morphologies depending on activated Akt 显示文摘Uhrbom L Dai C Celestino JC 2002Cancer Res2002,62,19:1
11Ink4a-Arf loss cooperates with KRas activation in astrocytes and neural progenitors to generate glioblastomas of various morphologies depending on activated Akt显示文摘Uhrbom L Dai C Celestino JC 0,,19:1
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