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19篇 您的检索式：作者名="Stanislas S"

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		题名	作者	年代	出处	被引量
	1	Noninvasive In Vivo Liver Fibrosis Evaluation Using Supersonic Shear Imaging: A Clinical Study on 113 Hepatitis C Virus Patients显示文摘	éric Bavu Jean-Luc Gennisson Mathieu Couade Jeremy Bercoff Vincent Mallet Mathias Fink Anne Badel Ana?s Vallet-Pichard Bertrand Nalpas Micka?l Tanter Stanislas Pol	2011	Ultrasound in Medicine & Biology2011，，9：	3
	2	A proterozoic-rift origin for the structure and the evolution of the cratonic Congo basin显示文摘	Etienne K K Stanislas S M N Francis L	2011	EPSL2011，304，1：	1
	3	Vaccination of chronic hepatitis B virus carriers with preS2/S envelope protein is not associated with the emergence of envelope escape mutants显示文摘	PATRICK S STANISLAS P FLORIANNE G	2001	J Gen Virol2001，82，：	1
	4	The organization of near-wall turbulence: a comparison between boundary layer SPIV data and channel flow DNS data显示文摘	Herpin S Stanislas M Soria J	2010	Journal of Turbulence2010，11，47：	1
	5	Localization of the Ulnar Nerve During Percutaneous Wiring of Supracondylar Fractures in Children显示文摘	MICHAEL S P STANISLAS MJC	1996	Injury1996，27，：	1
	6	Efficient replication of primary or culture hepatitis C virus isolates in human liver slices: A relevant ex vivo model of liver infection显示文摘	Sylvie Lagaye Hong Shen Bertrand Saunier Michelina Nascimbeni Jesintha Gaston Pierre Bourdoncle Laurent Hannoun Pierre‐Philippe Massault Ana?s Vallet‐Pichard Vincent Mallet Stanislas Pol	2012	Hepatology2012，，3：	1
	7	Factors in the surgical management of primary eccrine porocarcinoma: prognostic histo- logical factors can guide the surgical procedure显示文摘	Belin E Ezzedine K Stanislas S	2011	Br J Dermatol2011，165，5：	1
	8	Nonalcoholic Fatty Liver Disease and HIV Infection显示文摘	Ana?s Vallet-Pichard Vincent Mallet Stanislas Pol	2012	Semin Liver Dis2012，，02：	1
	9	Impaired expression of the peroxisome proliferator–activated receptor alpha during hepatitis C virus infection显示文摘	Sébastien Dharancy Mathilde Malapel Gabriel Perlemuter Tania Roskams Yang Cheng Laurent Dubuquoy Philippe Podevin Filoména Conti Valérie Canva David Philippe Luc Gambiez Philippe Mathurin Jean-Claude Paris Kristina Schoonjans Yvon Calmus Stanislas Pol Joh	2005	Gastroenterology2005，，2：	1
	10	Iridoids from Caryopteris x clandonensis 显示文摘	Hannedouche S Stanislas E Fourast6 I	2000	Phytochemistry2000，54，：	1
	11	Treatment of hepatitis C virus genotype 3‐infection显示文摘	Stanislas Pol Ana?s Vallet‐Pichard Marion Corouge	2014	Liver Int2014，，：	1
	12	Sources of mathematical thinking : behavioral and hrainimaging evidence显示文摘	Dehaene Stanislas Spelke Elizabeth S Philippe Pinel	1999	Science1999，284，：	1
	13	Acquired protein S deficiency leads to obliterative portal venopathy and to compensatory nodular regenerative hyperplasia in HIV-infected patients显示文摘	Vincent O Mallet Aditi Varthaman Dominique Lasne Jean-Paul Viard Hervé Gouya Delphine Borgel Sébastien Lacroix-Desmazes Stanislas Pol	2009	AIDS2009，，12：	1
	14	Nodular regenerative hyperplasia is a new cause of chronic liver disease in HIV-infected patients显示文摘	Vincent Mallet Pierre Blanchard Virginie Verkarre Ana?s Vallet-Pichard Hélène Fontaine Caroline Lascoux-Combe Stanislas Pol	2007	AIDS2007，，2：	1
	15	Noninvasive In Vivo Liver Fibrosis Evaluation Using Supersonic Shear Imaging: A Clinical Study on 113 Hepatitis C Virus Patients显示文摘	éric Bavu Jean-Luc Gennisson Mathieu Couade Jeremy Bercoff Vincent Mallet Mathias Fink Anne Badel Ana?s Vallet-Pichard Bertrand Nalpas Micka?l Tanter Stanislas Pol	2011	Ultrasound in Medicine & Biology2011，，9：	1
	16	The organization of near-wall turbulence:A comparison between boundary layer SPIV data and channel flow DNS data显示文摘	HERPIN S STANISLAS M SORIA J		0，，47：	1
	17	The organization of near-wall turbulence: A comparison between boundary layer SPIV data and channel flow DNS data 显示文摘	Herpin S Stanislas M Soria J	2010	Journal of Turbulence2010，11，47：	1
	18	Comparative genomic hybridization detects frequent over representation of chromosomal material from 3q26,8q24,and 20q13 in human ovarian carcinomas显示文摘	Gonosuke S Juan P Stanislas du M	1997	Genes Chromosomes Cancer1997，20，：	1
	19	Pathological evolution of hepatitis C virus-“Healthy carriers”显示文摘AIM: To determine factors associated with fibrosis progression in hepatitis C virus (HCV)-infected patients without signifi cant initial pathological lesions. METHODS: Seventy six untreated HCV-infected pa-tients with initially normal liver as defi ned by a Knodell score≤3,with 2 liver biopsies and detectable HCV-RNA were included. Markers of fibrosis progression were assessed. RESULTS: Median duration of infection and time between paired biopsies was 13 (95% CI: 1-28) and 4 (95% CI: 2-16) years respectively. Alanine-transaminase (ALT) activity was normal in 43.4% of cases. 50% demonstrated progression of the necro-in? ammation and 34% of fi brosis after a median time evolution of 4 years (95% CI: 2-16). The median dif-ference in the necro-inflammation and fibrosis score between biopsies was low,1.5 and 0.0 respectively. Univariate analysis showed there was no difference between fibrosis activity or evolution according to genotype or viral load. A higher fibrosis progression (P = 0.03) was observed in patients with body mass index (BMI) > 25. Fibrosis progression correlated with the time interval between biopsies (P = 0.01). A sig-nifi cant progression of activity (1.7 vs 0.4,P < 0.05) or fi brosis (0.9 vs 0.0,P < 0.01) was observed in patients with elevated ALT. There was a signifi cant correlation between activity progression and fi brosis progression (P = 0.003). Multivariate analysis demonstrated that fi brosis progression was associated with elevated ALT,BMI > 25 and the time interval between 2 biopsies. CONCLUSION: There is no fibrosis progression in 66% of patients without signifi cant initial histopatho-logical lesion. Fibrosis progression is associated with elevated ALT and BMI > 25.	Rodolphe Sobesky Pascal Lebray Bertrand Nalpas Anas Vallet-Pichard Hélène Fontaine Jean-Luc Lagneau Stanislas Pol	2008	World Journal of Gastroenterology2008，14，24：	0

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