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| 1 | K-ras^G12V transformation leads to mitochondrial dysfunction and a metabolic switch from oxidative phosphorylation to glycolysis显示文摘增加的氧气的 glycolysis 和氧化应力是癌症房间新陈代谢的重要特征,但是内在的生物化学、分子的机制留下逃犯。用四圜素可诱导的模型,我们显示出 K 地岬 G12V 原因 mitochondrial 机能障碍,导致反应的氧的减少的呼吸,提高的 glycolysis,和增加的产生种类。K 地岬蛋白质与线粒体被联系,并且导致呼吸的链建筑群的快速的抑制 -- 我和在由影响 cyclosporin 敏感的渗透的 mitochondrial transmembrane 潜力的减少转移毛孔。而且, K 地岬 在 vitro 的 G12V 表示提高转变房间的能力允许新陈代谢的改编到高 glycolytic 新陈代谢在 vivo 形成肿瘤。我们的学习建议 mitochondrial 机能障碍的那正式就职是由 K 地岬 G12V 在癌症房间引起新陈代谢的变化和 ROS 应力,并且支持肿瘤开发。 | Yumin Hu Weiqin Lu Gang Chen Peng Wang Zhao Chen Yan Zhou Marcia Ogasawara Dunyaporn Trachootham Li Feng Helene Pelicano Paul J Chiao Michael J Keating Guillermo Garcia-Manero Peng Huang | 2012 | Cell Research2012，22，2： | 6 |
| 2 | Effect of rubrene:P3HT bilayer on photovoltaic performance of perovskite solar cells with electrodeposited ZnO nanorods显示文摘Improved photovoltaic performance of perovskite solar cells is demonstrated through the synergistic effect of electrodeposited ZnO nanorods and rubrene:P3HT bilayer as electron and hole-transporting layers,respectively. Highly crystalline ZnO nanorods were obtained by electrochemical deposition in a chloride medium. Additionally, rubrene interlayer was able to passivate or cover the grain boundaries of perovskite film effectively that led to reduced leakage current. A perovskite solar cell optimized with ZnO nanorods and rubrene:P3HT bilayer achieved a maximum efficiency of 4.9% showing reduced hysteresis behavior compared with the device having P3HT as the only hole-transporting layer. The application of longer nanorods led to better perovskite infiltration and shorter charge carrier path length. These results highlight the potential of electrodeposited ZnO nanorods and rubrene:P3HT bilayer as charge selective layers for efficient perovskite solar cells. | Christian Mark Pelicano Hisao Yanagi | 2018 | Journal of Energy Chemistry2018，27，2： | 4 |
| 3 | Inhibition of glycolysis in cancer cells:a novel strategy to overcome drug resistance associated with mitochondrial respiratory defect and hypoxia显示文摘 | Xu RH Pelicano H Zhou Y | 2005 | Cancer Res2005，65，2： | 1 |
| 4 | Retinoic acid and interferon signaling cross talk in normal and RA-resistant APL cells 显示文摘 | Chelbi Alix MK Pelicano L | 1999 | Leukenia1999，13，8： | 1 |
| 5 | The circadian gene Period2 plays an important role in tumor suppression and DNA damage response in vivo显示文摘 | Fu L Pelicano H Liu J | 2002 | Cell2002，111，1： | 1 |
| 6 | The circadian gene Period2 plays an important role in tumor suppression and DNA dam- age response in vivo显示文摘 | Fu L Pelicano H Liu J | 2002 | Cell2002，111，1： | 1 |
| 7 | The circadian gene Period2 plays an important role in tumor suppression and DNA damage re- sponse in vivo显示文摘 | Fu L Pelicano H Liu J | 2002 | Ce112002，111，7： | 1 |
| 8 | Selective killing of oncogenically transformed cells through a ROS-mediated mechanism by β-phenylethyl isothiocyanate显示文摘 | Dunyaporn Trachootham Yan Zhou Hui Zhang Yusuke Demizu Zhao Chen Helene Pelicano Paul J. Chiao Geetha Achanta Ralph B. Arlinghaus Jinsong Liu Peng Huang | 2006 | Cancer Cell2006，，3： | 1 |
| 9 | ROS stress in cancer cells and therapeutic implications显示文摘 | Helene Pelicano Dennis Carney Peng Huang | 2004 | Drug Resistance Updates2004，，2： | 1 |
| 10 | Glycolysis inhibition for anticancer treatment显示文摘 | Pelicano H Martin DS Xu RH | | 0，，34： | 1 |
| 11 | Resistance to virus infection conferred by the interferon-induced promyelocytic leukemia protein显示文摘 | Chelbi-Alix M K Quignon F Pelicano L | 1998 | J Virol1998，72，： | 1 |
| 12 | Resistance to virus infection conferred by the interferon-induced promyelocytic leukemia protein显示文摘 | CHELBI A QUIGNON F PELICANO L | 1998 | J Virol1998，72，2： | 1 |
| 13 | The Circadian Gene Period2 Plays an Important Role in Tumor Suppression and DNA Damage Response In Vivo显示文摘 | Loning Fu Helene Pelicano Jinsong Liu Peng Huang Cheng Chi Lee | 2002 | Cell2002，，1： | 1 |
| 14 | The circadian gene Period2plays an important role in tumor suppression and DNA damage response in vivo显示文摘 | Fu L Pelicano H Liu J | 2002 | Cell2002，111，1： | 1 |
| 15 | The circadian gene period2 plays an important role in tumor suppression and DNA damage response in vivo 显示文摘 | Fu L Pelicano H Liu J | 2002 | Cell2002，111，1： | 1 |
| 16 | Inhibition of mitochondrial respiration:a novel strategy to enhance drug-induced apoptosis in human leukemia cells by a reactive oxygen species-mediated mechanism显示文摘 | Pelicano H Feng L Zhou Y | 2003 | J Biol Chem2003，278，37： | 1 |
| 17 | Mitochondrial dysfunction in some triple-negative breast cancer cell lines: role of roTOR path- way and therapeutic potential 显示文摘 | Pelicano H Zhang W Liu J | 2014 | Breast Cancer Res2014，16，5： | 1 |
| 18 | Mitochondrial dysfunction and reactive oxygen species imbalance promote breast Cancer ceil motility through a CXCL14-mediated mechanism显示文摘 | Pelicano H Lu W Zhou Y | | 0，，06： | 1 |
| 19 | The circadian gene Period2 plays an important role in tumor suppression and DNA damage response in vlvo显示文摘 | Fu L Pelicano H Liu J | 2002 | Cell2002，111，1： | 1 |
| 20 | The circadian gene period2 plays an important role in tumor suppression and DNA damage response in vivo显示文摘 | FU L Pelicano H LIU J | 2002 | Cell2002，111，1： | 1 |
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