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| 1 | The RAGE axis in early diabetic retinopathy显示文摘 | Barile GR Pachydaki SI Tari SR Lee SE Donmoyer CM Ma W | 2005 | Invest Ophthalm ol Vis Sci2005，46，8： | 1 |
| 2 | The RAGE axis in early diabetic retinopathy显示文摘 | Barile GR Pachydaki SI Tari SR | 2005 | Invest Ophthalmol Vis Sci2005，46，8： | 1 |
| 3 | The RAGE axis in early diabetic retinopathy显示文摘 | Barile G R Pachydaki S I Tari S R | 2005 | Invest Ophthalmol Vis Sci2005，46，8： | 1 |
| 4 | Surgical management and ultrastructural study of choroidal neovascularization in punctate inner choroidopathy after bevacizumab 显示文摘 | Pachydaki SI Jakobiee FA Bhat P Sobrin L Michaud NA Ses- hart SV | 2012 | J Ophthalmic Inflamm Infect2012，2，1： | 1 |
| 5 | The RAGE axis in early diabetic retinopathy显示文摘 | Barile GR Pachydaki SI Tari SR | 2005 | Invest Ophthalmol Vis Sci2005，46，8： | 1 |
| 6 | Efficacy of laparoscopic subtotal hysterectomy in the management of menorrhagia: 400 consecutive case显示文摘 | Erian J Hassan M Pachydakis A | 2008 | B JOG2008，115，6： | 1 |
| 7 | The RAGE axis in early diabetic retinopathy显示文摘 | Barile GR Pachydaki Sl Tari SR | | 0，，： | 1 |
| 8 | The RAGE axis in early diabetic retinopathy显示文摘 | Barile GR Pachydaki SI Tari SR | 2005 | Invest Ophthalmol Vis Sci2005，46，8： | 1 |
| 9 | Phenolypic features of patients with NR2E3 mutations显示文摘 | Pachydaki SI Klaver CC Barbazetto IA | 2009 | Arch Ophthalmol2009，127，1： | 1 |
| 10 | The RAGE axis in early diabetic retinopathy 显示文摘 | Barile GR Pachydaki SI Tari SR Lee SE Donmoyer CM Ma W | 2005 | Invest Ophthalmol Vis Sci2005，46，8： | 1 |
| 11 | Efficacy of laparoscopic subtotal hysterectomy in the management of menorrhagia:400 consecutive cases显示文摘 | Erian J Hassan M Pachydakis A Chandakas S Wissa I Hil N | | 0，，： | 1 |
| 12 | The RAGE axis in early diabetic retinopathy 显示文摘 | Barile GR Pachydaki SI Tari SR | 2005 | Invest Ophthalmol Vis Sci2005，46，8： | 1 |
| 13 | Photodynamic therapy and combination treatments显示文摘 | Pachydaki S Sobrin L Miller JW | 2007 | Int Ophthalmol Clin2007，47，1： | 1 |
| 14 | 增殖性玻璃体视网膜病变细胞凋亡的信号传导途径研究显示文摘目的 :探讨增殖性玻璃体视网膜疾病细胞凋亡的信号传导途径 ,以寻求新的药物治疗途径。方法 :2 3例增殖性玻璃体视网膜病变 (PVR) ,增殖性糖尿病性视网膜病变 (PDR) ,黄斑裂孔 (MH)及黄斑前膜 (MP)的视网膜前膜 (epiretinalmem brane,ERM )由玻璃体切割术中取得。细胞凋亡的情况由terminaldeoxynucleotidyltransfrase dUTP nickendlabeling(TUNEL法 )进行评估。Caspase 3及PARP的表达由特异性抗体抗活性Caspase 3和抗P 85片段的PARP检测。Cytokeratin与抗活性的Caspase 3双染色法进行凋亡细胞类型的鉴别。结果 :大多数发生凋亡的细胞为RPE细胞 ,而凋亡细胞与抗活性Caspase 3和抗P 85片段的PARP表达增加相关。细胞凋亡的数目与发生慢性视网膜脱离 (>2个月 )的病例有关 ,但凋亡系数 (apopto sisindex ,AI)在两组间无显著性差异 (1 4 4 2 9vs 3 2 2 86 ,P =0 1877)。PVR ,PDR ,MP各组的凋亡系数分别为 2 32 5 % ,3 4 2 % ,5 5 % ,P值分别为PPVR&PDR>0 1(0 16 85 ) ,PPDR&MP>0 1(0 5 380 ) ,PPVR&MP>0 1(0 8333)。结论 :此项研究发现细胞凋亡在PVR、PDR、MH及MP发病中的重要调节作用。诱导Caspase | 张新媛 Gaetano Barile Sophia Pachydaki William Schiff Janet Sparrow Stanley Chang | 2003 | 眼科2003，12，6： | 0 |
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