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35篇 您的检索式:作者名="POKREISZ P"
    题名 作者 年代 出处 被引量
1Cytochrome P450 epoxygenase gone function in hypoxic pulmonary, vasoconstriction and pulmonary vascular remodeling显示文摘Pokreisz P Fleming I Kiss L 2006Hypertension2006,47,4:1
2Sustained endothelial progenitor cell dysfunction after chronic hypoxia-induced pulmonary hypertension显示文摘MARSBOOM G POKREISZ P GHEYSENS O 2008Stem Cells2008,26,4:1
3Sustained endothelial progenitor cell dysfunction after chronic hypoxia-induced pulmonary hypertension显示文摘MARSBOOM G POKREISZ P GHEYSENS O 2008Stem Cells2008,26,4:1
4Cardiomyocyte - spe-cific overexpression of nitric oxide synthase 3 improves left ven-tricular performance and reduces compensatory hypertrophy aftermyocardial infarction 显示文摘Janssens S Pokreisz P Schoonjans L 2004Circ Res2004,94,9:1
5Pressure Overload - induced Right Ventricular Dysfunction and Remodeling in Experimental Pulmonary Hypertension: the Right Heart Revisited显示文摘Pokreisz P Marsboom G Janssens S 2007Eur Heart J Suppl2007,9,3:1
6Sustained endothelial progenitor cell dysfunction after chronic hypoxiainduced pulmonary hypertension显示文摘Marsboom G Pokreisz P Gheysens O 2008Stem Cells2008,26,4:1
7Aerosol gene transfer with inducible nitric oxide synthase reduces hypoxic pulmonary hypertension and pulmonary remodeling in rats显示文摘Budts W Pokreisz P Nong Z 2000Circulation2000,102,:1
8Sustained endothelial progenitor cell dysfunction after chronic hypoxia-induced pulmonary hypertension显示文摘Marsboom G Pokreisz P Gheysens O 2008Stem Cells2008,26,4:1
9Cardiomyocyte specific overexpreysion of nitric oxide synthase 3 improves left ventricuiar performance and reduces compensatory hypertrophy after myo- cardial infarction显示文摘Janssens S Pokreisz P 2004Circ Res2004,94,9:1
10Cardiomyocyte-specific overexpression of nitric oxide synthase 3 improves left ventricular performance and reduces compensatory hypertrophy after myocardial infarction显示文摘Janssens S Pokreisz P Schoonjans L 2004Circ Res2004,94,9:1
11Cardiomyocyte-specific overexpression of nitric oxide synthase 3 improves left ventricular performance and reduces compensatory hypertrophy after myocardial infarction显示文摘Janssens S Pokreisz P Schoonijans L 2004Circ Res2004,94,9:1
12Sustained endothelial progenitor cell dysfunction after chronic hypoxia-induced pulmonary hypertension 显示文摘Marsboom G Pokreisz P Gheysens O 2008Stem Cells2008,26,4:1
13Aerosol gene transfer with inducible nitric oxide synthase reduces hypoxic pulmonary hypertension and pulmonary vascular remodeling in rats显示文摘Budts W Pokreisz P Nong Z 2000Circulation2000,102,23:1
14Sustained en- dothelial progenitor cell dysfunction after chronic hypoxi- a-induced pulmonary hypertension显示文摘Marsboom G Pokreisz P Gheysens O 2008Stem Cells2008,26,4:1
15Ventricular phosphodiesterase-5 expression is increased in patients with advanced heart failure and contributes to adverse ventricular remodeling after myocardial infarction in mice显示文摘Pokreisz P Vandenwijngaert S Bito V 0,,:1
16Effect of adenovirus-mediated gene transfer of nitric oxide synthase on vascular reactivity of rat isolated pulmonary arteries 显示文摘Jiang L Quarck R Janssens S Pokreisz P Demedts M Delcroix M 2006Pflugers Arch2006,452,2:1
17Ventricularphosphodiesterase-5 expression is increased in patients with advancedheart failure and contributes to adverse ventricular remodeling aftermyocardial infarction in mice显示文摘Pokreisz P Vandenwijngaert S Bito V 2009Circulation2009,119,3:1
18Functional impairment of endothelial progenitor cells in hypoxic pulmonary hypertension 显示文摘Marsboom G Pokreisz P Vermeersch P 2006Euro Heart J2006,27,:1
19Ventricular phosphodiesterase-5 expression is increased in patients with advanced heart failure and contributes to adverse ventricular remodeling after myocardial infarction in mice显示文摘Pokreisz P Vandenwijngaert S Bito V 2009Circulation2009,119,:1
20Car diomyoeyte specific overexpression of nitric oxide synthase 3 improves left ventrieular performance and reduces eompensa tory hypertrophy after myocardial infarction显示文摘JANSSENS S POKREISZ P SCHOENJANS L 2004Circ Res2004,94,:1
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