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21篇 您的检索式:作者名="Kohlmeier S"
    题名 作者 年代 出处 被引量
1Occult vitamin D deficiency in postmenopausal US women with acute hip fracture显示文摘LeBoff M Kohlmeier L Hurwitz S 1999JAMA1999,281,16:1
2Ligand-induced muopioid receptor endocytosis and recycling in enteric neurons显示文摘MINNIS JG PATIERNO S KOHLMEIER SE 20102010,,:1
3Wnt signaling and skeletal development显示文摘Liu F Kohlmeier S Wang CY 0,,06:1
4Occult vita min D defficiency in postmenopausal US women with acute hip fracture 显示文摘LeBoff MS Kohlmeier L Hurwitz S 1999JAMA1999,281,:1
5Taking the fungal highway: mobilization of pollutant-degrading bacteria by fungi 显示文摘KOHLMEIER S SMITS T H M FORD R M 2005Environ Sci Tecbnol2005,39,12:1
6Epidemiologic evidence of role of carotenoids in cardiovascular disease prevention显示文摘KOHLMEIER L HASTINGS S H 1995Am J Clin Nutr1995,,:1
7Wnt signaling and skeletal development显示文摘Liu F Kohlmeier S Wang C Y 2008Cell Signal2008,20,6:1
8Tea and cancer prevention:an evaluation of the epidemiologic literature显示文摘Kohlmeier L Weterings K G Steck S 1997Nutr Cancer1997,27,1:1
9CXCR3 directs anti-gen-specific effector CD4+T cell migration to the lung during parain-fluenza virus infection显示文摘Kohlmeier J E Cookenham T Miller S C 2009Immunol2009,183,7:1
10Wnt signaling and skeletal development显示文摘Liu F Kohlmeier S Wang CY 2008Cell Signal2008,20,6:1
11Mesomorphic block molecules: semiperfluorinated 1,3,5-triazine derivatives exhibiting lamellar, columnar, and cubic mesophases显示文摘KOHLMEIER A jANIETZ D DIELE S 2006Chem Mater2006,18,6:1
12Activation phenotype, rather than central- or effector-memory phenotype, predicts the recall efficacy of memory CD8^+ T cells 显示文摘Hikono H Kohlmeier JE Takamura S 2007J Exp Med2007,204,:1
13Taking the fungal highway: mobilization of pollutant-degrading bacteria by fungi 显示文摘Kohlmeier S Smits T H M Ford R M 2005Environ Sci Technol2005,39,12:1
14Wnt signaling and skeletal development显示文摘LIU F KOHLMEIER S WANG C Y 2008Cell Signal2008,20,6:1
15Occult vita-min D defficiency in postmenopausal US women with acutehip fracture 显示文摘LeBoff MS Kohlmeier E Hurwitz S 1999JAMA1999,281,:1
16Ligand-inducedmuopioid receptor endocytosis and recycling in enteric neurons显示文摘Minnis JG Patiemo S Kohlmeier SE 2003Neuroscience2003,119,1:1
17Occult vitamin D deficiency in postmenopausal US women with acute hip fracture显示文摘LeBoff MS Kohlmeier L Hurwitz S 1999JAMA1999,281,16:1
18Anti gen-specific memory regulatory CD4^+ Foxp3^+ T cells control memory responses to influenza virus infection 显示文摘Brincks E L Roberts A D Cookenham T Sell S Kohlmeier J E Blaekman M A Woodland D L 2013J Immunol2013,190,7:1
19Prenatal nicotine alters development of the laterodorsal tegmentum:Possible role for attention-deficit/hyperactivity disorder and drug dependence显示文摘As we cycle between the states of wakefulness and sleep,a bilateral cholinergic nucleus in the pontine brain stem,the laterodorsal tegmentum(LDT),plays a critical role in controlling salience processing,attention,behavioral arousal,and electrophysiological signatures of the sub-and microstates of sleep.Disorders involving abnormal alterations in behavioral and motivated states,such as drug dependence,likely involve dysfunctions in LDT signaling.In addition,as the LDT exhibits connectivity with the thalamus and mesocortical circuits,as well as receives direct,excitatory input from the prefrontal cortex,a role for the LDT in cognitive symptoms characterizing attention-deficit/hyperactivity disorder(ADHD)including impulsivity,inflexibility,and dysfunctions of attention is suggested.Prenatal nicotine exposure(PNE)is associated with a higher risk for later life development of drug dependence and ADHD,suggesting alteration in development of brain regions involved in these behaviors.PNE has been shown to alter glutamate and cholinergic signaling within the LDT.As glutamate and acetylcholine are major excitatory mediators,these alterations would likely alter excitatory output to target regions in limbic motivational circuits and to thalamic and cortical networks mediating executive control.Further,PNE alters neuronal development and transmission within prefrontal cortex and limbic areas that send input to the LDT,which would compound effects of differential processing within the PNE LDT.When taken together,alterations in signaling in the LDT are likely to play a role in negative behavioral outcomes seen in PNE individuals,including a heightened risk of drug dependence and ADHD behaviors.Filip S Polli Kristi A Kohlmeier 2022World Journal of Psychiatry2022,12,2:1
20Writ signaling and skeletal development显示文摘Liu F Kohlmeier S Wang CY 2008Cellular Signalling2008,20,:1
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