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17篇 您的检索式:作者名="Grandage"
    题名 作者 年代 出处 被引量
1When less is more:a practical approach to searching for evidence - based answers显示文摘Grandage K Slawson DC Shanghnessy A 2002J Med Libr Assoc2002,90,3:1
2PI3-kinase/Akt is constitutively active in primary acute myeloid 1 eukaemia cells and regulates survival and chemoresis-tance via NF-kappaB, Mapkinase and p53 pathways显示文摘Grandage VL Gale RE Linch DC 2005Leukemia2005,19,4:1
3PI3-kinase/Akt is constitutively active in primary acute myeloid leukaemia cells and regulates survival and chemoresistance via NF-kappaB, Mapkinase and p53 pathways显示文摘Grandage VL Gale RE Linch DC 2005Leukemia2005,19,4:1
4p13-kinase/Akt is constitutivelyactive in primary acute myeloid leukaemia cells and regulates survival and chemoresistance via NF-kappaB,Mapkinase and p53 pathways显示文摘Grandage VL Gale RE Linch DC 2005Leukemia2005,19,4:1
5Constitutive activation of the Wnt/β-catenin signalling pathway in acute myeloid leukaemia 显示文摘Simon M Grandage VL Khwaja A 2005Oncogene2005,24,14:1
6Constitutive activation of the Wnt/beta-catenin signalling pathway in acute myeloid leukaemia 显示文摘Simon M Grandage VL Linch DC 2005Oncogene2005,24,14:1
7PI3-kinase/Akt is constitutively active in primary acute myeloid leukaemia cells and regulates survival and chemoresistance via NF-kappaB, Mapkinase and p53 pathways显示文摘Grandage VL Gale RE Linch DC 2005Leukemia2005,19,4:1
8A selective inhibitor of the p110deha isoform of PI 3-kinase inhibits AML cell proliferation and survival and increases the cytotoxic effects of VP16 显示文摘Billottet C Grandage VL Gale RE 2006On- cogene2006,25,50:1
9Constitutive activation of the Wnt/beta-catenin signalling pathway in acute myeloid leukaemia 显示文摘Simon M Grandage VL Linch DC 2005Oncogene2005,24,14:1
10Constitutive activation of the Wnt/β-catenin signalling pathway in acute myeloid leukaemia显示文摘Simon M Grandage VL Linch DC 2005Oncogene2005,24,2:1
11PI3-kinase/Akt is constitutively active in primary acute myeloid leukemia cells and regulates survival and chemo resistance via NF-kappa B,MAPK and p53pathways显示文摘Grandage V L Gale R E Linch D C 0,,:1
12PI3 - kinase/Akt is constitutively active in primary acute myeloid leukaemia cells and regulates survival and chemoresistance via NF - kappaB, Mapkinase and p53 pathways 显示文摘Grandage VL Gale RE Linch DC 2005Leukemia2005,19,4:1
13PI3-kinase/Akt is constitutively active in primary acute myeloid leukemia cells and regulates survival and chemoresistanee via NF-kappaB, MAPK and p53 pathways 显示文摘Grandage VL Gale RE Linch DC 2005Leukemia2005,19,4:1
14PI32kinase /Akt isconstitutively active in primary acute myeloid leukaemia cells and regulates survival and chemoresistance via NF-kappaB,Mapkinase and p53 pathways显示文摘Grandage VL Gale RE Linch DC 0,,:1
15When less is more:a practical approach to searching for evidence-based answers显示文摘Grandage KK Slawson DC Shaughnessy AF 2002Bull Med Libr Assoc2002,90,3:1
16PI3-ki- nase/Akt is constitutively active in primary acute myeloid leukaemia cells and regulates survival and chemoresistance via NF-kappaB, Mapkinase and p53 pathways显示文摘Grandage VL Gale RE Linch DC 2005Leukemia2005,19,4:1
17Constitutive activation of the Wnt/13-catenin signalling pathway in acute myeloid lenkaemia显示文摘Simon M Grandage VL Linch DC 2005Oncogene2005,24,14:1
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