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12篇 您的检索式:作者名="FOLMER F"
    题名 作者 年代 出处 被引量
1Biomedical research tools from the seabed显示文摘Folmer F Houssen W E Scott R H etal 2007Curt Opin Drug Discov Devel2007,10,:1
2DNA primers for amplification of mitochondrial cytochrome c oxidase subunit I from diverse metozoan invertebrates显示文摘Folmer O Black M Hoen F 1994Mol Mar Biol Biot1994,,3:1
3Anticancer effects of bioactive berry compounds显示文摘FOLMER F BASAVARAJU U JASPARS M 2014Phytoehem Rev2014,13,1:1
4DNA primers for a mp- lification of mitochondrial cytochrome coxidase subunit I from diverse metazoan invertebrates显示文摘Folmer O Black M Hoen F 1994Mol Mar Biol Biotechnol1994,3,5:1
5Interaction of the replication terminator protein of Bacillus subtilis with DNA probed by NMR spectroscopy 显示文摘HASTINGS A F OTTING G FOLMER R H 2005Biochem Biophys Res Commun2005,335,2:1
6The Arabidopsis HOBBIT gene encodes a CDC27 homolog that links the plant cell cycl to progression of cell differentiation显示文摘BLILOU I FRUGIER F FOLMER S 2002Genes Development2002,16,10:1
7Removal of pesticides and other micropollutants with cellulose-acetate, polyamide and ultra-low pressure reverse osmosis membranes显示文摘Hofman J A M H Becrendonk E F Folmer H C 1997Desalination1997,113,23:1
8Marine natural products as targeted modulators of the transcription factor NF-κB显示文摘Folmer F Jaspars M Dicato M 0,,03:1
9NF-kappaB -inhibiting naphthopyrones from the Fijian echinoderm Comanthus parvicirrus显示文摘Folmer F Harrison WT Tabudravu JN 2008J Nat Prod2008,71,1:1
10Marine Natural Products Targeting Phospholipases Az 显示文摘FOLMER F JASPARS M SCHUMACHER M 2010Biochem Pharmacol2010,80,12:1
11DNA primers for amplification of mi- tochondrial cytochrome c oxidase subunit I from diverse metazoan inver- tebrates显示文摘Folmer O Black M Hoen F 1994MolMarBiol Biot1994,3,5:1
12Oxidative stress and labile plasmatic iron in anemic patients following blood therapy显示文摘AIM: To determine the plasmatic iron content and evaluate the oxidative stress(OS) markers in subjects receiving blood therapy.METHODS: Thirty-nine individuals with unspecified anemia receiving blood transfusions and 15 healthy subjects were included in the study. Anemic subjects were divided into three subgrou P:(1) those that received up to five blood transfusions(n = 14);(2) those that received from five to ten transfusions(n = 11); and(3) those that received more than ten transfusions(n = 14). Blood samples were collected by venous arm puncture and stored in tubes containing heparin. The plasma and cells were separated by centrifugation and subsequently used for analyses. Statistical analyses were performed using Kruskal-Wallis analysis of variance followed by Dunn's multiple comparison tests when appropriate.RESULTS: The eletrophoretic hemoglobin profiles of the subjects included in this study indicated that no patients presented with hemoglobinopathy. Labile plasmatic iron, ferritin, protein carbonyl, thiobarbituric acidreactive substances(TBARS) and dichlorofluorescein diacetate oxidation were significantly higher(P < 0.05), whereas total thiol levels were significantly lower(P < 0.05) in transfused subjects compared to controls. Additionally, the activity of catalase, superoxide dismutase and glutathione peroxidase were significantly lower in the transfused subjects(P < 0.05). Antioxidant enzyme activities and total thiol levels were positively correlated(P < 0.05), and negatively correlated with the levels of protein carbonyl and TBARS(P < 0.05). In contrast, protein carbonyl and TBARS were positively correlated(P < 0.05). Altogether, these data confirm the involvement of OS in patients following therapy with repeated blood transfusions.CONCLUSION: Our data reveal that changes in OS markers are correlated with levels of labile plasmatic iron and ferritin and the number of transfusions.Marília Sabo Fernandes Tatiana Tamborena Rissi Luisa Zuravski Juliana Mezzomo Carmen Regla Vargas Vanderlei Folmer Félix Alexandre Antunes Soares Vanusa Manfredini Mushtaq Ahmed Robson Luiz Puntel 2014World Journal of Experimental Medicine2014,4,3:0
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