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    题名 作者 年代 出处 被引量
1骨密度测量中精密度的重要性显示文摘骨密度测定 ,不论采取何特殊技术 ,即使严格地按照厂家建议的操作程序 ,也常常不能达到完美的重复性。必须确定每台骨密度仪不同骨骼部位的精确度。精密度 ,如标准差平均方根或变异系数平均方根 ,被用来确定骨密度的变化 ,即精密度决定最小显著性变化值和随访需要的至少时间间隔。除非确定了精密度 ,否则就不能确定任何水平的统计可信度最小显著性变化值 。Sydney Lou Bonnick C.Conrad Johnston Michael Kleerekoper Robert Lindsay Paul Miller Louis Sherwood Ethel Siris 周琦 程晓光 2002中国骨质疏松杂志2002,8,1:12
2Sphincter of Oddi dysfunction:Psychosocial distress correlates with manometric dyskinesia but not stenosis显示文摘AIM:To compare postcholecystectomy patients with Sphincter of Oddi(SO)dyskinesia and those with normal SO motility to determine the psychosocial distress,gender and objective clinical correlates of dyskinesia,and contrast these findings with comparisons between SO stenosis and normal SO motility.METHODS:Within a cohort of seventy-two consecutive postcholecystectomy patients with suspected SO dysfunction,manometric assessment identified subgroups with SO dyskinesia(n=33),SO stenosis(n=18)and normal SO motility(n=21).Each patient was categorized in terms of Milwaukee Type,sociodemographic status and the severity of stress-coping experiences.RESULTS:Logistic regression revealed that in combination certain psychological,sociodemographic and clinical variables significantly differentiated SO dyskinesia,but not SO stenosis,from normal SO function.Levels of psychosocial stress and of coping with this stress(i.e.anger suppressed more frequently and the use of significantly more psychological coping strategies)were highest among patients with SO dyskinesia,especially women.Higher levels of neuroticism(the tendency to stressproneness)further increased the likelihood of SO dyskinesia.CONCLUSION:A motility disturbance related to psychosocial distress may help to explain the finding of SO dyskinesia in some postcholecystectomy patients.Ethelle Bennett Peter Evans John Dowsett John Kellow 2009World Journal of Gastroenterology2009,15,48:4
3Effectiveness of a microabrasion technique using 16% HCL with manual application on fluorotic teeth: A series of studies显示文摘BACKGROUND Dental fluorosis is caused by excessive fluoride ingestion during tooth formation.As a consequence,there is a higher porosity on the enamel surface,which causes an opaque look.AIM The aim of this study was to identify a dental intervention to improve the smile in patients with tooth fluorosis.Additional aims were to relate the stain size on fluorotic teeth with the effectiveness of stain removal,enamel loss and procedure time using a manual microabrasion technique with 16%hydrochloric acid(HCL).METHODS An experimental study was carried out on 84 fluorotic teeth in 57 adolescent patients,33 females and 24 males,with moderate to severe fluorosis.The means,standard deviations and percentages were analyzed using nonparametric statistics and ArchiCAD 15 software was used for the variables including stain size and effectiveness of stain removal.RESULTS The average enamel loss was 234μm and was significantly related to the procedure time categorized as 1-4 min and 4.01-6 min,resulting in a P>0.000.The microabrasion technique using 16%HCL was effective in 90.6%of patients and was applied manually on superficial stains in moderate and severe fluorosis.Procedure time was less than 6 min and enamel loss was within the acceptable range.CONCLUSION Microabrasion is a first-line treatment;however,the clinician should measure the average enamel loss to ensure that it is within the approximate range of 250μm in order to avoid restorative treatment.Martina Nevárez-Rascón Nelly Molina-Frechero Edith Adame Ethel Almeida Uriel Soto-Barreras Enrique Gaona Alfredo Nevárez-Rascón 2020World Journal of Clinical Cases2020,8,4:2
4Diosgenin stimulates osteogenic activity by increasing bone matrix protein synthesis and bone-specific transcription factor Runx2 in osteoblastic MC3T3-E1 cells显示文摘Ethel H. Alcantara Mee-Young Shin Ho-Yong Sohn Youn-Moon Park Taewan Kim Jae-Hwan Lim Hyung-Jin Jeong Soon-Tae Kwon In-Sook Kwun 2011The Journal of Nutritional Biochemistry2011,,11:2
5Platelet‐associated antibodies, cellular immunity and FCGR3a genotype influence the response to rituximab in immune thrombocytopenia显示文摘Nichola Cooper Roberto Stasi Susanna Cunningham‐Rundles Ethel Cesarman Janice G. McFarland James B. Bussel 2012Br J Haematol2012,,4:2
6Ca^2+ -induced apoptosis through calcineurin dephosphorylation of BAD 显示文摘Wang HG Pathan N Ethell IM 1999Science1999,284,:1
7Increased Anti-M llerian hormone serum concertrations in prepubertal daughters of women with polycystic ovary syndrome(PCOS)显示文摘Teresa S P Ethel C Manuel M 2006The Journal of Clinical Endocrinology Metabolism2006,91,8:1
8Information and Decision Making: Scanning the Environment for Strategic Advantage 显示文摘Auster Ethel Choo Chun Wei 2001Library Trends2001,34,2:1
9Involvement of the pancreas in AIDS: a prospective study of 109 post-mortems显示文摘Ethel Z Chehter Maria Antonieta Longo Antonio A Laudanna Maria Irma S Duarte 2000AIDS2000,,13:1
10Autoantibodies Against the Fibrinolytic Receptor, Annexin A2, in Cerebral Venous Thrombosis显示文摘Gabriela Cesarman-Maus Carlos Cantú-Brito Fernando Barinagarrementeria Rosario Villa Elba Reyes Jorge Sanchez-Guerrero Katherine A. Hajjar Ethel Garcia Latorre 2011Stroke2011,,2:1
11Ca2+ Induced Apoptosis through caclcineurin dephosphorylation of BAD显示文摘 Pathan N Ethell IM 1999Science1999,284,5412:1
12Ephrin-B2-induced cleavage of EphB2 receptor ismediated by matrix metalloproteinases to trigger cellrepulsion 显示文摘LIN K T SLONIOWSKI S ETHELL D W 2008J Biol Chem2008,283,28:1
13Comparison of second cancer risks from brachytherapy and external beam therapy after uterine corpus cancer显示文摘Stefan L Ethel G S ElaineR 2010Cancer Epidemiol Biomarkers Prey2010,19,2:1
14A delicate balance: role ofMMP-9 in brain development and pathophysiology of neurode-velopmental disorders 显示文摘Reinhard SM Razak K Ethell IM 2015Front Cell Neurosci2015,9,:1
15Release of dissolved organic matter in a tropical strain of Synura petersenii (Chrysophyceae) grown under high irradiances显示文摘Vieira A A H Novelli Ethel L B Rodrigues Ney L 1998Phycologia1998,37,5:1
16Third-line and rescue therapy for refractory Helicobacter pylori infection: A systematic review显示文摘BACKGROUND Due to increasing resistance rates of Helicobacter pylori(H.pylori)to different antibiotics,failures in eradication therapies are becoming more frequent.Even though eradication criteria and treatment algorithms for first-line and second-line therapy against H.pylori infection are well-established,there is no clear recommendation for third-line and rescue therapy in refractory H.pylori infection.AIM To perform a systematic review evaluating the efficacy and safety of rescue therapies against refractory H.pylori infection.METHODS A systematic search of available rescue treatments for refractory H.pylori infection was conducted on the National Library of Medicine’s PubMed search platform based on Preferred Reporting Items for Systematic Reviews and Meta-Analyses guidelines.Randomized or non-randomized clinical trials and observational studies evaluating the effectiveness of H.pylori infection rescue therapies were included.RESULTS Twenty-eight studies were included in the analysis of mean eradication rates as rescue therapy,and 21 of these were selected for analysis of mean eradication rate as third-line treatment.For rifabutin-,sitafloxacin-,levofloxacin-,or metronidazole-based triple-therapy as third-line treatment,mean eradication rates of 81.6%and 84.4%,79.4%and 81.5%,55.7%and 60.6%,and 62.0%and 63.0%were found in intention-to-treat(ITT)and per-protocol(PP)analysis,respectively.For third-line quadruple therapy,mean eradication rates of 69.2%and 72.1%were found for bismuth quadruple therapy(BQT),88.9%and 90.9%for bismuth quadruple therapy,three-in-one,Pylera®(BQT-Pylera),and 61.3%and 64.2%for non-BQT)in ITT and PP analysis,respectively.For rifabutin-,sitafloxacin-,levofloxacin-,or metronidazole-based triple therapy as rescue therapy,mean eradication rates of 75.4%and 78.8%,79.4 and 81.5%,55.7%and 60.6%,and 62.0%and 63.0%were found in ITT and PP analysis,respectively.For quadruple therapy as rescue treatment,mean eradication rates of 76.7%and 79.2%for BQT,84.9%and 87.8%for BQT-Pylera,and 61.3%and 64.2%for non-BQT were found in ITT and PP analysis,respectively.For susceptibility-guided therapy,mean eradication rates as third-line and rescue treatment were 75.0%in ITT and 79.2%in PP analysis.CONCLUSION We recommend sitafloxacin-based triple therapy containing vonoprazan in regions with low macrolide resistance profile.In regions with known resistance to macrolides or unavailability of bismuth,rifabutin-based triple therapy is recommended.Pedro Vieira de Moraes Andrade Yan Mosca Monteiro Ethel Zimberg Chehter 2023World Journal of Gastroenterology2023,29,2:1
17Rethinking teaching nursing homes:Potential for improving long-term care显示文摘Mathy D Mezey RN Ethel L 2008The Gerontologist2008,48,:1
18Maneb potentiates paraquat neurotoxicit, by inducing key Bcl-2 family members显示文摘Fei Q Ethell I)W 2008J Neurchem2008,105,6:1
19An early CT-diagno- sis-based treatment strategy for invasive fungal infection in al- logeneic transplant recipients using caspofungin first line: an effective strategy with 10w mortality显示文摘Dignan FL Evans SO Ethell ME 2009Bone Marrow Transplant2009,44,1:1
20Molecular mechanisms of dendritic spine development and remodeling显示文摘Ethell IM Pasquale EB 2005Prog Neurobiol2005,75,:1
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