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| 1 | Postpartum depression:A systematic review of the genetics involved显示文摘Postpartum depression is one of the most prevalent psychopathologies. Its prevalence is estimated to be between 10% and 15%. Despite its multifactorial etiology, it is known that genetics play an important role in the genesis of this disorder. This paper reviews epidemiological evidence supporting the role of genetics in postpartum depression(PPD). The main objectives of this review are to determine which genes and polymorphisms are associated with PPD and discuss how this association may occur. In addition, this paper explores whether these genes are somehow related to or even the same as those linked to Major Depression(MD). To identify gaps in the current knowledge that require investigation, a systematic review was conducted in the electronic databases Pub Med, LILACS and Sci ELO using the index terms 'postpartum depression' and 'genetics'. Literature searches for articles in peerreviewed journals were made until April 2014. PPD was indexed 56 times with genetics. The inclusion criteria were articles in Portuguese, Spanish or English that were available by institutional means or sent by authors upon request; this search resulted in 20 papers. Genes and polymorphisms traditionally related to MD, which are those involved in the serotonin, catecholamine, brain-derived neurotrophic factor and tryptophan metabolism, have been the most studied, and some have been related to PPD. The results are conflicting and some depend on epigenetics, which makes the data incipient. Further studies are required to determine the genes that are involved in PPD and establish the nature of the relationship between these genes and PPD. | Tiago Castro e Couto Mayra Yara Martins Brancaglion António Alvim-Soares Lafaiete Moreira Frederico Duarte Garcia Rodrigo Nicolato Humberto Corrêa Regina Amélia Lopes P Aguiar Henrique Vitor Leite | 2015 | World Journal of Psychiatry2015，5，1： | 5 |
| 2 | Perception of the esthetic impactof mandibular incisor extraction treatment on laypersons,dentalprofessionals,and dental students显示文摘 | Pithon MM Santos AM Couto FS | 2012 | Angle Orthod2012，82，4： | 1 |
| 3 | The role of ADP-ribosyla- tion in regulating DNA double-strand break repair显示文摘 | Pears CJ Couto AM Wang HY | 2012 | Cell Cy- cle2012，11，1： | 1 |
| 4 | Calpain inhibitor 2 prevents axonal degeneration of opos- sum optic nerve fibers 显示文摘 | Araujo Couto L Sampaio Narciso M Hokoc JN Blanco Martinez AM | 2004 | J Neurosci Res2004，77，3： | 1 |
| 5 | Whiceblood cellcountab-normalities and infections in one-year follow-up of124patients with SLE显示文摘 | Dias AM Couto MC Duarte CC et a1 | 2009 | Ann N Y Acad Sci2009，17，3： | 1 |
| 6 | White blood cell count abnormalities and infections in one-year follow-up of 124 pa- tients with SLE显示文摘 | Dias AM do Couto MC Duarte CC | 2009 | Ann N Y Acad Sci2009，1173，： | 1 |
| 7 | White blood cell count abnormalities and infections in one-year follow-up of 124 patients with SLE显示文摘 | Dias AM do Couto MCj Duarte CC | 2009 | Ann N YAcad Sci2009，1173，： | 1 |
| 8 | Mammalian target of rapamycin pathway mutations cause hemimegalencephaly and focal cortical dysplasia显示文摘 | D'Gama AM Geng Y Couto JA | 2015 | Ann Neurol2015，77，4： | 1 |
| 9 | Histological aspects of rhinosinusal polyps显示文摘 | Couto LG Fernades AM Brandso DF | 2008 | Revista Brasileira de Otorrinolaringologia2008，74，2： | 1 |
| 10 | White blood cell count abnor- malities and infections in one-year follow-up of 124 patients with SLE 显示文摘 | Dias AM do Couto MC Duarte CC | 2009 | Ann N Y Acad Sci2009，1173，： | 1 |
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