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		题名	作者	年代	出处	被引量
	1	The gut microbiome as therapeutic target显示文摘	Patrice D. Cani Nathalie M. Delzenne	2011	Pharmacology and Therapeutics2011，，2：	2
	2	应用激光捕获显微切割分析深部子宫内膜异位症和位置正常子宫内膜的芳香酶及17β-羟类固酮脱氢酶2型mRNA表达显示文摘Objective: To investigate mRNA expression of aromatase and 17β - hydroxysteroid dehydrogenase type 2 (17β HSD2) in epithelial and stromal cells from eutopic and ectopic endometrium of patients with deep endometriosis. Design: Prospective study. Setting: University hospital. Patient(s): Patients with deep endometriosis and fertile women with macroscopically normal pelvic cavities. Intervention(s): During surgery,30 endometrial and 16 endometriotic samples were obtained from 30 patients with deep endometriosis. Control endometrial samples were obtained from 24 fertile women with macroscopically normal pelvic cavities who underwent laparoscopic tubal ligation or reversal of tubal sterilization. Epithelial cells and stromal cells from endometrial or endometriotic tissues were microdissected using laser capture microdissection. Main Outcome Measure(s): Expression levels of aromatase and 17β HSD2 mRNA in microdissected epithelial and stromal cells were determined using quantitative real- time reverse transcriptase polymerase chain reaction. Result(s): Aromatase mRNA expression was significantly higher in epithelial cells than in stromal cells in both eutopic and ectopic endometrium obtained from endometriosis patients. In the ectopic endometrium of 8 patients (8/16, 50% ), 17β HSD2 expression was not detected in either epithelial or stromal cells. In eutopic endometrium from endometriosis patients, 17β HSD2 expression in epithelial cells was significantly increased during the early, middle, and late secretory phases compared with the late proliferative phase, whereas no significant cyclical difference was detected in control endometrium. Conclusion(s): Local estrogen concentration may be much higher in epithelial cells than in stromal cells in deep endometriotic tissue.	Matsuzaki S. Canis M. Pouly J.-L. 张旸	2006	世界核心医学期刊文摘(妇产科学分册)2006，2，6：	1
	3	Selective increases of bifidobacteria in gut microflora improve high-fat-diet-induced diabetes in mice through a mechanism associated with endotoxaemia显示文摘	P. D. Cani A. M. Neyrinck F. Fava C. Knauf R. G. Burcelin K. M. Tuohy G. R. Gibson N. M. Delzenne	2007	Diabetologia2007，，11：	1
	4	Selective increases of bifidobacteria in gut microflora improve high-fat-diet-induced diabetes in mice through a mechanism associated with endotoxaemia显示文摘	P. D. Cani A. M. Neyrinck F. Fava C. Knauf R. G. Burcelin K. M. Tuohy G. R. Gibson N. M. Delzenne	2007	Diabetologia2007，，11：	1
	5	Harnessing the beneficial properties of adipogenic microbes for improving human health显示文摘	N. V. Dhurandhar L. Geurts R. L. Atkinson L. Casteilla K. Clement P. Gerard M. Vijay‐Kumar J. H. Nam M. Nieuwdorp G. Trovato T. I. A. S?rensen A. Vidal‐Puig P. D. Cani	2013	Obes Rev2013，，9：	1
	6	Selective increases of bifidobacteria in gut microflora improve high-fat-diet-induced diabetes in mice through a mechanism associated with endotoxaemia显示文摘	P. D. Cani A. M. Neyrinck F. Fava C. Knauf R. G. Burcelin K. M. Tuohy G. R. Gibson N. M. Delzenne	2007	Diabetologia2007，，11：	1
	7	Selective increases of bifidobacteria in gut microflora improve high-fat-diet-induced diabetes in mice through a mechanism associated with endotoxaemia显示文摘	P. D. Cani A. M. Neyrinck F. Fava C. Knauf R. G. Burcelin K. M. Tuohy G. R. Gibson N. M. Delzenne	2007	Diabetologia2007，，11：	1
	8	Selective increases of bifidobacteria in gut microflora improve high-fat-diet-induced diabetes in mice through a mechanism associated with endotoxaemia显示文摘	P. D. Cani A. M. Neyrinck F. Fava C. Knauf R. G. Burcelin K. M. Tuohy G. R. Gibson N. M. Delzenne	2007	Diabetologia2007，，11：	1
	9	Menses recovery and fertility after artery embolization for PPH: a single-center retrospective observational study显示文摘	G. Gaia P. Chabrot L. Cassagnes A. Calcagno D. Gallot R. Botchorishvili M. Canis G. Mage L. Boyer	2009	European Radiology2009，，2：	1
	10	重度子宫内膜异位症患者的在位内膜环氧化酶-2表达高于对照组	Matsuzaki S. Canis M. Pouly J.-L. 张剑萍	2005	世界核心医学期刊文摘(妇产科学分册)2005，0，4：	0

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