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19篇 您的检索式:作者名="CASSARINO D"
    题名 作者 年代 出处 被引量
1High-throughput pipeline for de-novo assembly and drug resistance mutations iden- tification frmn next-generation sequencing viral data of residual diagnostic samples显示文摘Cassarino TG Frampton D Sugar R 2015BioRxiv2015,,03:1
2Extending Group Technology to The Identification and The Analysis of Enterprises Net-works显示文摘Villa A Cassarino I Antonelli D 2007International Journal of Production Research2007,45,17:1
3Spinal adrenal cortical adenoma with oncocytic features: report of the first intramedullary case and review of the literature显示文摘Cassarino D S Santi M Arruda A 2004Int J Surg Pathol2004,12,3:1
4Up-regulated Dicer ex- pression in patients with cutaneous melanoma 显示文摘Ma Z Swede H Cassarino D 2011PLoS One2011,6,20:1
5Up-regula-ted Dicer expression in patients with cutaneous melano-ma显示文摘MA Z SWEDE H CASSARINO D 2011PLoS One2011,,:1
6Inhibition by R(+) or S(-) pramipexole of caspase activation and cell death induced by methylpyridinium ion or beta amyloid peptide in SH-SY5Y neuroblastoma显示文摘Abramova N A Cassarino D S Khan S M Painter T W Bennett J P Jr 2002J Neurosci Res2002,67,:1
7An evaluation of the role of mitochondria in neurodegenerative disease:mitochondrial mutations and oxidative pathology,protective nuclear response,and cell death in neurodegeneration显示文摘CASSARINO D S BENNETF J P J R 1999Brain Res Rev1999,29,:1
8Cybrids in Alzheimer's disease:a cellular model of the disease?显示文摘Swerdlow R H Parks J K Cassarino D S 1997Neurology1997,49,4:1
9The Parkinsonian neurotoxin MPP + opens the mitochondrial permeabicity transition pore and release cytochrome Cin isolated mitochondria via an oxidative mechanism显示文摘Cassarino D S Parks J K Parker W D Jr 1999Biochim Biophys Acta1999,1453,1:1
10Up-regulated Dicer expression in patients with cutaneous melanoma显示文摘Ma Z Swede H Cassarino D 0,,:1
11Up-regulated Dicer ex- pression in patients with cutaneous melanoma显示文摘Ma Z Swede H Cassarino D 2011PoLS One2011,6,20:1
12An evaluation of the role of mitochondria in neurodegenerative diseases: mitochondrial mutations and oxidative pathology, protective nuclear responses, and cell death in neurodegeneration显示文摘Cassarino D S Bennett J P Jr 1999Brain Res Rev1999,29,1:1
13The parkinsonian neurotoxin MPP+ opens the mitochondrial permeability transition pore and releases cytochrome C in isolated mitochondrial via an oxidative mechanism显示文摘CASSARINO D S PARKS J K PARKER W D 1999Biochim Biophys Acta1999,1453,1:1
14Up-regulated Dicerl expression in patients with cutaneous melanoma显示文摘Ma Z Swede H Cassarino D 2011PLoS One2011,6,20:1
15Interaction among mitochondria, mitogen-activated protein kinase, and nuclear factor-κB in cellular models of Parkinson's disease显示文摘 Halvorsen E M Swerdlow R H 2000J Neurochem2000,74,:1
16The parkinsonian neurotoxin MPP^+ opens the mitochondrial permeability transition pore and releases cytochrome C in isolated mitochondria via an oxidative mechanism显示文摘Cassarino D S Parks J K Parker W D 1999Biochim Biophys Acta1999,1453,1:1
17Spinal adrenal cortical adenoma with oncocytic features: report ofthe first intramedullary case and review of the literature显示文摘Cassarino D S Santi M Arruda A 2004Int J Surg Pathol2004,12,:1
18The parkinsonian neurotoxin MPPl opens the mitochondrial permeability transition pore and releases cytochrome c in isolated mitochondria via an oxidative mechanism显示文摘CASSARINO D S PARKS J K PARKER W D 1999Biochim Biophys Acta1999,1453,12:1
19The parkinsonian neurotoxin MPP+ opens the mitochondrial permeability transition pore and releases cytochrome C in isolated mitochondrail via an oxidative mechanism显示文摘Cassarino D S Parks J R Pareker W D 1999Biochem Biophys Acta1999,1453,1:1
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