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24篇 您的检索式：作者名="Baartscheer"

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	题名	作者	年代	出处	被引量
1	Energy-dependent transport of calcium to the extracelluar space during acute ischemia of the rat heart显示文摘	Schumacher C A Baartscheer A Coronel R	1998	J Mol Cardiol1998，30，：	1
2	i and the driving force of the Na ^+/Ca^2 + -exchanger in heart failure 显示文摘	Baartscheer A Schumacher CA Belterman CN	2003	Cardiovasc Res2003，57，15：	1
3	Chronic inhibition of Na^+/H^+ exchanger attenuates cardiac hypertrophy and prevents cellular remodeling in heart failure 显示文摘	Baartscheer A Schumacher CA Borren MM	2005	Cardiovasc Res2005，65，1：	1
4	Chronic inhibition of Na^+/H^+ -exchanger attenuates cardiac hypertrophy and prevents cellular remodeling in heart failure显示文摘	Baartscheer A Schumacher CA van Borren MM	2005	Cardiovasc Res2005，65，1：	1
5	i and the driving force of the Na+/Ca^2+-exchanger in heart failure 显示文摘	Baartscheer A Schumacher CA Belterman CNW	2003	Cardiovasc Res2003，57，：	1
6	Contribution of Sodium Channel Mutations to Bradycardia and Sinus Node Dysfunction in LQT3 Families显示文摘	Marieke W. Veldkamp Ronald Wilders Antonius Baartscheer Jan G. Zegers Connie R. Bezzina Arthur A.M. Wilde	2003	Circulation Research: Journal of the American Heart Association2003，，9：	1
7	Contribution of sodium channel mutations to bradycardia and sinus node dysfunction in LQT3 families显示文摘	Wilders R Baartscheer A	2003	Circ Res2003，92，9：	1
8	Chronicinhibition of the Na+/H+-exchanger causes regression ofhypertrophy,heart failure,and ionic and electrophysiologicalremodelling显示文摘	Baartscheer A Hardziyenka M Schumacher CA	2008	Br J Pharmacol2008，154，6：	1
9	Increased Na^+/H^+ - exchange activity is the cause of increased i and underlies disturbed calci- um handling in the rabbit pressure and volume overload heart failure model 显示文摘	Baartscheer A Schumacher C A van Borren M M	2003	Cardiovasc Res2003，57，4：	1
10	Increased Na +/H + - exchange activity is the cause of increased i and underlies disturbed calcium handling in the rabbit pressure and volume overload heart failure model 显示文摘	Baartscheer A Schumacher CA van Borren MM	2003	Cardiovasc Res2003，57，4：	1
11	SR calcium handling and calcium after-transients in a rabbit model of heart failure显示文摘	Baartscheer A Schumacher CA Belterman CNW	2003	Cardiovasc Res2003，58，1：	1
12	Ionic mechanism of delayed aflerdepolarizations in ventricular cells isolated from human end-stage failinghearts显示文摘	Verkerk AO Veldkamp MW Baartscheer A	2002	Circulation2002，104，22：	1
13	i and the driving force of the Na/Ca-exchanger in heart failure显示文摘	Baartscheer A Schumacher CA Belterman CNW	2003	Cardiovascular Research2003，57，4：	1
14	Contribution of sodium channel mutations to bradycardia and sinus node dysfunction in LQT3 families显示文摘	Veldkamp MW Wilders R Baartscheer A		0，，：	1
15	Osmotic changes and transsarcolemmal ion transport during total ischaemia of isolated rat ventricular myocytes显示文摘	FIOLET JW SCHUMACHER CA BAARTSCHEER A	1993	Basic Res Cardiol1993，88，5：	1
16	Contribution of NHE-1 to cell length shortening of normal and failing rabbit cardiac myocytes显示文摘	van Borren Mm Zegers JG Baartscheer A	2006	J Mol Cell Cardiol2006，41，4：	1
17	Ionic mechanism of delayed after depolarizations in ventricular cells isolated from human end stage failing hearts显示文摘	Verkerk AO Veldkamp MW Baartscheer A	2001	Circulation2001，104，22：	1
18	Increased Na^+/H^+ -exchange activity is the cause of increased i and underlies disturbed calcium handling in the rabbit pressure and volume overload heart failure model 显示文摘	Baartscheer A Schumacher CA Van BMM	2003	Cardiovasc Res2003，57，4：	1
19	Energy-dependent transport of calcium to the extracellular space during acute ischemia of the rat heart显示文摘	Baartscheer A Coronel R	1998	J Mol Cell Cardiol1998，30，：	1
20	Contribution of sodium channel mutations to bradycardia and sinus node dysfunction in LQT3 families 显示文摘	Veldkamp MW Wilders R Baartscheer A	2003	Circ Res2003，92，9：	1

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