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34篇 您的检索式:作者名="BENEDICTO I"
    题名 作者 年代 出处 被引量
1Expression of pituitary tumor-transforming gene 1(PTTG1)/securin in hepa-titis B virus(HBV)-associated liver diseases:evidence for an HBV X protein-mediated inhibition of PTTG1 ubiquitination and degradation显示文摘Molina-Jimenez F Benedicto I Murata M 0,,03:1
2Increased circulating proinflammatory cytokines and Th17 lymphocytes in Hashimoto' s thyroiditis显示文摘Figueroa-Vega N Alfonso-Pérez M Benedicto I 2010J Clin Endocrinol Metab2010,95,2:1
3In- creased circulating pro-inflammatory cytokines and Th17 lym- phocytes in Hashimoto's thyroiditis显示文摘Figueroa-Vega N Alfonso-Perez M Benedicto I 2010J Clin Endocrinol Metab2010,95,:1
4Increased circulating proinflammatory cytokines and Th17 lymphocytes in hashimoto's thyroiditis显示文摘FIGUEROA VEGA N ALFONSO PEREZ M BENEDICTO I 2010Clin Endocrinol Metab2010,95,2:1
5Epithelial-to- mesenchymal transition of peritoneal mesothelial cells is regulated by an ERK/NF-kappa B/Snaill pathway 显示文摘Strippoli R Benedicto I P6rez Lozano ML 2008Dis Model Mech2008,1,45:1
6Increased circulating proinflammatory cytokines and Th 17 lymphocytes in Hashimotors thyroiditis显示文摘Figueroa-Vega N Alfonso-P6rez M Benedicto I 2010J Clin Endocrinol Metab2010,95,2:1
7p38 maintains E-cadherin expression by modulating TAK1 - NF-kappa B during epithelial-to-mesenchymal transition 显示文摘STRIPPOLI R BENEDICTO I FORONDA M 2010J Cell Sci2010,123,24:1
8Inhibition of transtbr- ming growth factor-activated kinase 1 ( TAK1 ) blocks and reverses epi- thelial to mesenchymal transition of mesothelial cells 显示文摘Strippoli R Benedicto I Perez Lozano ML 2012PIoS Otle2012,7,31:1
9Increased circulating pro-inflammatory cytokines and Thl7 lymphocytes in Hashimoto's thyroiditis 显示文摘Figueroa-Vega N Alfonso-Perez M Benedicto I 2010J Clin Endocrinol Metab2010,95,2:1
10Neuropilin-1: A feasible link between liver pathologies and COVID-19显示文摘The severe acute respiratory syndrome coronavirus 2(SARS-CoV-2)pandemic has a tremendous impact on the health of millions of people worldwide.Unfortunately,those suffering from previous pathological conditions are more vulnerable and tend to develop more severe disease upon infection with the new SARS-CoV-2.This coronavirus interacts with the angiotensin-converting enzyme 2 receptor to invade the cells.Recently,another receptor,neuropilin-1(NRP-1),has been reported to amplify the viral infection.Interestingly,NRP-1 is expressed in nonparenchymal liver cells and is related to and upregulated in a wide variety of liver-related pathologies.It has been observed that SARS-CoV-2 infection promotes liver injury through several pathways that may be influenced by the previous pathological status of the patient and liver expression of NRP-1.Moreover,coronavirus disease 2019 causes an inflammatory cascade called cytokine storm in patients with severe disease.This cytokine storm may influence liver sinusoidal-cell phenotype,facilitating viral invasion.In this review,the shreds of evidence linking NRP-1 with liver pathologies such as hepatocellular carcinoma,liver fibrosis,nonalcoholic fatty liver disease and inflammatory disorders are discussed in the context of SARS-CoV-2 infection.In addition,the involvement of the infection-related cytokine storm in NRP-1 overexpression and the subsequent increased risk of SARS-CoV-2 infection are also analyzed.This review aims to shed some light on the involvement of liver NRP-1 during SARSCoV-2 infection and emphasizes the possible involvement this receptor with the observed liver damage.Aitor Benedicto Iñigo García-Kamiruaga Beatriz Arteta 2021World Journal of Gastroenterology2021,27,24:1
11In-creased circulating pro-inflammatory cytokines and Thl7lymphocytes in Hashimoto's thyroiditis 显示文摘Figueroa-Vega N Alfonso-Perez M Benedicto I 2010J Clin Endo-crinol Metab2010,95,2:1
12Increased circulating pro-inflammatory cytokines and Thl7 lymphocytes inHashimoto's thyroiditis 显示文摘Figueroa-Vega N Alfonso-P6rez M Benedicto I 2010J Clin Endocrinol Metab2010,95,2:1
13The tight junction-associated protein oceludin is required for a postbinding step in hepatitis C virus entry and infection 显示文摘BENEDICTO I MOLINA-JIMENEZ F BARTOSCH B 2009J Virol2009,83,16:1
14In- creased circulating pro - inflammatory cytokines and Thl7 lympho- eytes in Hashimoto' s thymiditis 显示文摘Figueroa - Vega N Alfonso - Perez M Benedicto I 2010J Clin Endocrinol Metab2010,95,2:1
15Increased cir- culating pro-inflammatory cytokines and Thl7 lymphocytes in Hashim- oto' s thyroiditis 显示文摘Figueroa-Vega N Alfonso-Perez M Benedicto I 2010J Clin Endocrinol Metab2010,95,2:1
16Increased circulating pro-inflammatory cytokines and Thl7 lymphocytes in Hashimoto's thyroiditis 显示文摘Figueroa Vega N Alfonso-P6rez M Benedicto I 2010J Clin Endocrinol Metab2010,95,2:1
17Increasedcirculating pro -inflammatory cytokines and Thl7 lymphocytes inHashimoto's thyroiditis 显示文摘Fi gueroa -Vega N Alfonso -Perez M Benedicto I 2010J Clin Endocrinol Metab2010,95,2:1
18Inhibition of transforming growth factor-activated kinase 1(TAK1)blocks and reverses epithelial to mesenchymal transition of mesothelial cells显示文摘Strippoli R Benedicto I Perez Lozano ML 2012PLoS One2012,7,31:1
19Increased circulating pro-inflammatory cytokines and Th17 lymphocytes in Hashimoto's thyroiditis显示文摘Figueroa-Vega N Alfonso-Perez M Benedicto I 2010J Clin Endocr Metab2010,95,2:1
20In- creased circulating pro-inflammatory cytokines and Thl7 lymphocytes in Hashimoto's thyroiditis显示文摘Figueroa-Vega N Alfonso-Perez M Benedicto I 2010J Clin En- docrinol Metab2010,95,2:1
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